Bernát S I, Metz E, Gonda F, Torday A
Magyar Honvédség Központi Honvédkórház, Budapest 3. Belgyógyászat-Kardiológia.
Orv Hetil. 1998 Sep 27;139(39):2319-21.
The authors examined the rate of myocardial infarction at young age (under 50 years old) in group of 36 patients with a pathological gene (P1 A2) and in control group (55 healthy persons). They couldn't find difference between the results of two groups. After that they created a group from patients with "low risk rate" for myocardial infarction. In this group the rate of pathological gene is twofold, than in the control group. It is so seem, that in special cases of myocardial infarction the P1 A2 gene is the most important risk factor to produce the pathological function of thrombocytes and so to produce a higher incidence of arterial thrombosis (for example in coronaries).
作者研究了36例携带病理基因（P1 A2）的患者以及对照组（55名健康人）中年轻人（50岁以下）的心肌梗死发生率。他们未发现两组结果之间存在差异。此后，他们从心肌梗死“低风险率”患者中创建了一个组。在该组中，病理基因的发生率是对照组的两倍。似乎在心肌梗死的特殊情况下，P1 A2基因是导致血小板病理功能从而导致动脉血栓形成（例如冠状动脉血栓形成）发生率升高的最重要风险因素。
