Murphy B G, Macklem P T
Respir Physiol. 1976 Oct;28(1):65-74. doi: 10.1016/0034-5687(76)90085-2.
The transmission of forces between the visceral and parietal pleural is modelled as being mediated by two distinct pathways: (a) a contact pressure (Pcon) acting on an area of contact (Acon) and (2) a pleural liquid pressure (Pliq) acting on the area of liquid anfractuosities (Aliq). Summation of the forces yields a new equation relating pleural surface pressure (Ppl) to Pcon, Acon, Pliq, and Aliq. A complete solution of the equation is restricted by present limitations on measurement technology. However, assuming that the volume of pleural liquid is constant during tidal breathing, analysis of the model explains the findings that tidal swings in Pliq (deltaPliq) are sometimes greater than tidal swings in Ppl (deltaPpl). It also predicts that the influence of a change of lung elastic recoil on Pliq may be different in magnitude than its effect on Ppl. Therefore deltaPliq is not necessarily equivalent to deltaPpl.
（a）作用于接触面积（Acon）上的接触压力（Pcon），以及（2）作用于液体曲折面积（Aliq）上的胸膜液体压力（Pliq）。这些力的总和产生了一个将胸膜表面压力（Ppl）与Pcon、Acon、Pliq和Aliq相关联的新方程。该方程的完整解受到当前测量技术限制的约束。然而，假设在潮式呼吸期间胸膜液体的体积恒定，对该模型的分析解释了以下发现：Pliq的潮式波动（deltaPliq）有时大于Ppl的潮式波动（deltaPpl）。它还预测，肺弹性回缩变化对Pliq的影响在大小上可能与其对Ppl的影响不同。因此，deltaPliq不一定等同于deltaPpl。
