Identification of a novel CBFB-MYH11 fusion transcript in a patient with AML and inversion of chromosome 16. - Suppr | 超能文献

Identification of a novel CBFB-MYH11 fusion transcript in a patient with AML and inversion of chromosome 16.

作者信息

Springall F H, Lukeis R L, Tyrrell V, Joshua D E, Iland H J

出版信息

Leukemia. 1998 Dec;12(12):2034-5. doi: 10.1038/sj.leu.2401209.

DOI:10.1038/sj.leu.2401209

Abstract

摘要

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Identification of a novel CBFB-MYH11 fusion transcript in a patient with AML and inversion of chromosome 16.

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The NDE1 gene is disrupted by the inv(16) in 90% of cases with CBFB-MYH11-positive acute myeloid leukemia.在90%的CBFB-MYH11阳性急性髓系白血病病例中，NDE1基因被inv(16)破坏。

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Detection and quantification of CBFB/MYH11 fusion transcripts in patients with inv(16)-positive acute myeloblastic leukemia by real-time RT-PCR.通过实时逆转录聚合酶链反应检测和定量inv(16)阳性急性髓性白血病患者中的CBFB/MYH11融合转录本

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CBFB/MYH11 fusion transcripts in a case of acute myelogenous leukemia (M1) with partial deletion of the long arm of chromosome 16.16号染色体长臂部分缺失的急性髓性白血病（M1）病例中的CBFB/MYH11融合转录本

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Identification of a novel CBFB-MYH11 transcript: implications for RT-PCR diagnosis.一种新型CBFB-MYH11转录本的鉴定：对逆转录聚合酶链反应诊断的意义

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Detection of a novel CBFB-MYH11 fusion transcript in acute myeloid leukemia M1 with inv(16)(p13q22).在伴有inv(16)(p13q22)的急性髓系白血病M1中检测到一种新型CBFB-MYH11融合转录本。

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Novel CBFB-MYH11 fusion transcripts or reverse transcription-polymerase chain reaction artifacts?新型CBFB-MYH11融合转录本还是逆转录-聚合酶链反应假象？

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Inversion of chromosome 16 and uncommon rearrangements of the CBFB and MYH11 genes in therapy-related acute myeloid leukemia: rare events related to DNA-topoisomerase II inhibitors?治疗相关急性髓系白血病中16号染色体倒位及CBFB和MYH11基因的罕见重排：与DNA拓扑异构酶II抑制剂相关的罕见事件？

J Clin Oncol. 1998 May;16(5):1890-6. doi: 10.1200/JCO.1998.16.5.1890.

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Acute myeloid leukemia with t(16;16) (p13;q22) showing a new CBFB-MYH11 fusion transcript associated with an atypical leukemic blasts morphology.伴有 t(16;16)(p13;q22) 的急性髓系白血病，表现出一种新的 CBFB-MYH11 融合转录本，与非典型白血病原始细胞形态相关。

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引用本文的文献

1

CBFB-MYH11/RUNX1 together with a compendium of hematopoietic regulators, chromatin modifiers and basal transcription factors occupies self-renewal genes in inv(16) acute myeloid leukemia.CBFB-MYH11/RUNX1 与一系列造血调节因子、染色质修饰因子和基础转录因子一起，占据了 inv(16) 急性髓系白血病中的自我更新基因。

Leukemia. 2014 Apr;28(4):770-8. doi: 10.1038/leu.2013.257. Epub 2013 Sep 4.