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前交叉韧带重建术后“独眼巨人”病变的不同方面:多因素发病机制

Different aspects of the cyclops lesion following anterior cruciate ligament reconstruction: a multifactorial etiopathogenesis.

作者信息

Delincé P, Krallis P, Descamps P Y, Fabeck L, Hardy D

机构信息

Department of Traumatology and Orthopaedic Surgery, Centre Hospitalier Universitaire St. Pierre, Brussels, Belgium.

出版信息

Arthroscopy. 1998 Nov-Dec;14(8):869-76. doi: 10.1016/s0749-8063(98)70025-8.

Abstract

After anterior cruciate ligament (ACL) reconstruction using a patellar-tendon autograft, 65 patients underwent second-look arthroscopy in conjunction with hardware removal. In 23 patients, hypertrophic tissue was found in the anterior part of the knee. This tissue presented different aspects, from a well-synovialized nodule to a more disorganized fibrous tissue according to patients' complaints. Endoscopic resection of this offending tissue was generally sufficient to obtain a satisfactory result. In patients presenting a loss of extension, the notch frequently had to be enlarged. We have found a multifactorial pathogenesis to be likely: the nodule is a natural fibroproliferative tissue process originating either from drilling debris from the tibial tunnel or from remnants of the ACL stump and, more rarely, from broken graft fibers. Sometimes, when the graft is malpositioned, the scar tissue can result from repeated graft impingement on the notch at terminal extension. Formation of this aberrant tissue should be prevented by proper positioning of the graft, by enlargement of the narrowed notch in chronic cases, by using drills of increasing diameters to avoid production of osteocartilaginous fragments, by meticulous resection of all drilling debris and ACL remnants around the tibial tunnel, and by enlarging the notch roof if any contact with the graft is present when the knee is fully extended intraoperatively.

摘要

在使用髌腱自体移植物进行前交叉韧带(ACL)重建后,65例患者在取出内固定装置的同时接受了二次关节镜检查。在23例患者中,在膝关节前部发现了肥厚组织。根据患者的主诉,这种组织呈现出不同的形态,从滑膜化良好的结节到更杂乱的纤维组织。对这种有害组织进行内镜下切除通常足以获得满意的结果。对于出现伸直受限的患者,通常需要扩大髁间窝。我们发现其发病机制可能是多因素的:结节是一种自然的纤维增生性组织过程,其起源要么是来自胫骨隧道的钻孔碎屑,要么是ACL残端的残余物,更罕见的是来自断裂的移植物纤维。有时,当移植物位置不当,瘢痕组织可能是由于移植物在伸直末期反复撞击髁间窝所致。应通过正确放置移植物、在慢性病例中扩大狭窄的髁间窝、使用直径逐渐增大的钻头以避免产生骨软骨碎片、仔细切除胫骨隧道周围的所有钻孔碎屑和ACL残余物,以及在术中膝关节完全伸直时如果移植物有任何接触则扩大髁间窝顶来预防这种异常组织的形成。

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