Karmody A M, Powers S R, Monaco V J, Leather R P
Arch Surg. 1976 Nov;111(11):1263-8. doi: 10.1001/archsurg.1976.01360290097015.
We describe 31 patients in whom proximal lesions in the arterial tree were identified as probable sources of emboli causing the "blue toe" syndrome. This syndrome consists of acute digital ischemia caused by microembolization to the digital arteries from a proximal source via a patent arterial tree, as evidenced by an otherwise well-perfused foot. It is closely analogous to the transient ischemic attacks of the brain, and carries the same potential for serious tissue loss because of repeated embolic showers. The prompt delineation and eradication of the embolic source is of prime importance, in addition to restoration of arterial continuity. Along with the other well-known features of chronic severe ischemia, that is, rest pain, gangrene, etc, the "blue toe" syndrome is therefore an indication for limb salvage surgery.
我们描述了31例患者,其动脉树近端病变被确定为可能导致“蓝趾”综合征的栓子来源。该综合征由近端来源的栓子经通畅的动脉树微栓塞至指动脉引起急性手指缺血所致,足部其他部位血供良好可作为证据。它与脑短暂性缺血发作极为相似,且因反复的栓子脱落而具有同样严重组织丢失的可能性。除恢复动脉连续性外,迅速明确并消除栓子来源至关重要。因此,“蓝趾”综合征连同慢性重度缺血的其他众所周知的特征,即静息痛、坏疽等,都是肢体挽救手术的指征。