Swinney G R, Foster S F, Church D B, Malik R
Department of Veterinary Clinical Sciences, University of Sydney, New South Wales.
Aust Vet J. 1998 Nov;76(11):722-4. doi: 10.1111/j.1751-0813.1998.tb12299.x.
Two dogs developed a disabling gait abnormality characterised by stiffness. The abnormality was consistent with a diagnosis of myotonia secondary to hyperadrenocorticism. The first dog had iatrogenic hyperadrenocorticism, and its signs improved substantially after corticosteroid administration was gradually withdrawn. The second had pituitary-dependent hyperadrenocorticism, but myotonic signs progressed despite effective mitotane therapy. Procainamide administration reduced the myotonic stiffness in the second case.
