血红素加氧酶通过降解血红素抑制一氧化氮合酶：一氧化氮产生的负反馈调节机制。 - Suppr

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超能文献

Heme oxygenase inhibits nitric oxide synthase by degrading heme: a negative feedback regulation mechanism for nitric oxide production.

作者信息

Turcanu V, Dhouib M, Poindron P

机构信息

Department of Immunopharmacology, University Louis Pasteur, Strasbourg, France.

出版信息

Transplant Proc. 1998 Dec;30(8):4184-5. doi: 10.1016/s0041-1345(98)01385-2.

DOI:10.1016/s0041-1345(98)01385-2

PMID:9865340

Abstract

摘要

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Heme oxygenase inhibits nitric oxide synthase by degrading heme: a negative feedback regulation mechanism for nitric oxide production.血红素加氧酶通过降解血红素抑制一氧化氮合酶：一氧化氮产生的负反馈调节机制。

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Nitric oxide synthase inhibition by haem oxygenase decreases macrophage nitric-oxide-dependent cytotoxicity: a negative feedback mechanism for the regulation of nitric oxide production.血红素加氧酶对一氧化氮合酶的抑制作用降低了巨噬细胞一氧化氮依赖性细胞毒性：一种调节一氧化氮产生的负反馈机制。

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Inhibition of inducible nitric oxide synthase by peroxisome proliferator-activated receptor agonists: correlation with induction of heme oxygenase 1.过氧化物酶体增殖物激活受体激动剂对诱导型一氧化氮合酶的抑制作用：与血红素加氧酶1诱导的相关性

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Biphasic induction of heme oxygenase-1 expression in macrophages stimulated with lipopolysaccharide.脂多糖刺激巨噬细胞中血红素加氧酶-1表达的双相诱导

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Mechanism of increase of heme oxygenase activity induced by hemin in cultured pig alveolar macrophages.

Arch Biochem Biophys. 1979 Oct 15;197(2):607-17. doi: 10.1016/0003-9861(79)90285-6.

Nitric oxide and prostaglandin E2 participate in lipopolysaccharide/interferon-gamma-induced heme oxygenase 1 and prevent RAW264.7 macrophages from UV-irradiation-induced cell death.一氧化氮和前列腺素E2参与脂多糖/γ干扰素诱导的血红素加氧酶1的生成，并防止RAW264.7巨噬细胞发生紫外线照射诱导的细胞死亡。

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Erythropoietin controls heme metabolic enzymes in normal human bone marrow culture.促红细胞生成素在正常人骨髓培养中控制血红素代谢酶。

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[Heme oxygenase activity in the tissues of the vessels and heart of rats under co-administration of NO-synthase inhibitor and hemin chloride].[一氧化氮合酶抑制剂与氯化血红素共同给药时大鼠血管和心脏组织中的血红素加氧酶活性]

Ukr Biokhim Zh (1999). 2008 Mar-Apr;80(2):128-34.

In vitro and in vivo induction of heme oxygenase-1 in rat glial cells: possible involvement of nitric oxide production from inducible nitric oxide synthase.大鼠神经胶质细胞中血红素加氧酶-1的体外和体内诱导：诱导型一氧化氮合酶产生一氧化氮的可能作用。

Glia. 1998 Feb;22(2):138-48.

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Microglia are the major cellular source of inducible nitric oxide synthase during experimental herpes encephalitis.在实验性疱疹性脑炎期间，小胶质细胞是诱导型一氧化氮合酶的主要细胞来源。

J Neurovirol. 2008 May;14(3):229-38. doi: 10.1080/13550280802093927.

A set of genes previously implicated in the hypoxia response might be an important modulator in the rat ear tissue response to mechanical stretch.一组先前与缺氧反应有关的基因可能是大鼠耳部组织对机械拉伸反应的重要调节因子。

BMC Genomics. 2007 Nov 23;8:430. doi: 10.1186/1471-2164-8-430.

Heme oxygenase/carbon monoxide-biliverdin pathway down regulates neutrophil rolling, adhesion and migration in acute inflammation.血红素加氧酶/一氧化碳-胆绿素途径可下调急性炎症中中性粒细胞的滚动、黏附和迁移。

Br J Pharmacol. 2006 Oct;149(4):345-54. doi: 10.1038/sj.bjp.0706882. Epub 2006 Sep 4.

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