Correspondence re: S. Fulda et al., Betulinic acid triggers CD95 (Apo1/Fas)- and p53-independent apoptosis via activation of caspases in neuroectodermal tumors. Cancer Res., 57: 4956-4964, 1997.
作者信息
Rieber M, Rieber M S
出版信息
Cancer Res. 1998 Dec 15;58(24):5876-7.
PMID:9865749
Abstract
摘要
相似文献
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Correspondence re: S. Fulda et al., Betulinic acid triggers CD95 (Apo1/Fas)- and p53-independent apoptosis via activation of caspases in neuroectodermal tumors. Cancer Res., 57: 4956-4964, 1997.致函：S. 富尔达等人，《桦木酸通过激活神经外胚层肿瘤中的半胱天冬酶触发不依赖CD95（Apo1/Fas）和p53的细胞凋亡》。《癌症研究》，第57卷：4956 - 4964页，1997年。
Cancer Res. 1998 Dec 15;58(24):5876-7.
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Betulinic acid induces apoptosis through a direct effect on mitochondria in neuroectodermal tumors.桦木酸通过直接作用于神经外胚层肿瘤中的线粒体诱导细胞凋亡。
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Betulinic acid: a new chemotherapeutic agent in the treatment of neuroectodermal tumors.桦木酸：一种治疗神经外胚层肿瘤的新型化疗药物。
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Betulinic acid-induced programmed cell death in human melanoma cells involves mitogen-activated protein kinase activation.桦木酸诱导人黑色素瘤细胞程序性细胞死亡涉及丝裂原活化蛋白激酶激活。
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Betulinic acid promotes TRAIL function on liver cancer progression inhibition through p53/Caspase-3 signaling activation.桦木酸通过激活p53/半胱天冬酶-3信号通路促进TRAIL在抑制肝癌进展中的作用。
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Betulinic acid induces apoptosis in human neuroblastoma cell lines.
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