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The clinically isolated FIZ15 bacteriophage causes lysogenic conversion in Pseudomonas aeruginosa PAO1.

作者信息

Vaca-Pacheco S, Paniagua-Contreras G L, García-González O, de la Garza M

机构信息

Laboratorio de Genética, Escuela Nacional de Estudios Profesionales Iztacala, Universidad Nacional Autónoma de México, Apartado postal 314, Tlalnepantla, Edo. de Méx., 54090, México.

出版信息

Curr Microbiol. 1999 Apr;38(4):239-43. doi: 10.1007/pl00006794.

DOI:10.1007/pl00006794
PMID:10069861
Abstract

FIZ15 bacteriophage, from a human clinical isolate of Pseudomonas aeruginosa, causes lysogenic conversion in the P. aeruginosa strain PAO1. The prophage-conferred phenotypes are: (1) increased resistance to phagocytosis by mouse peritoneal macrophages; (2) increased resistance to killing by normal human serum, and (3) increased adhesion to human buccal epithelial cells. These phenotypes are related to the prophage-induced change at the level of its own bacterial receptor, which appears to be the O-antigen.

摘要

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