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Kupffer's cells modulate neutrophile activity by superoxide anion and tumor necrosis factor-delta in reperfusion injury of liver transplantation-mechanisms of radical generation and reperfusion injury after cold ischemia.

作者信息

Ohkohchi N, Shibuya H, Tsukamoto S, Sakurada M, Oikawa K, Terashima T, Satomi S

机构信息

Second Department of Surgery, Tohoku University School of Medicine, Sendai, Japan.

出版信息

Transplant Proc. 1999 Feb-Mar;31(1-2):1055-8. doi: 10.1016/s0041-1345(98)01902-2.

DOI:10.1016/s0041-1345(98)01902-2
PMID:10083472
Abstract
摘要

相似文献

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Kupffer's cells modulate neutrophile activity by superoxide anion and tumor necrosis factor-delta in reperfusion injury of liver transplantation-mechanisms of radical generation and reperfusion injury after cold ischemia.库普弗细胞在肝移植再灌注损伤中通过超氧阴离子和肿瘤坏死因子-δ调节中性粒细胞活性——冷缺血后自由基生成及再灌注损伤机制
Transplant Proc. 1999 Feb-Mar;31(1-2):1055-8. doi: 10.1016/s0041-1345(98)01902-2.
2
Activation of Kupffer cells and neutrophils for reactive oxygen formation is responsible for endotoxin-enhanced liver injury after hepatic ischemia.库普弗细胞和中性粒细胞被激活以形成活性氧，这是肝缺血后内毒素增强肝损伤的原因。
Shock. 1995 Jan;3(1):56-62.
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Modulation of ischemia-reperfusion-induced hepatic injury by Kupffer cells.库普弗细胞对缺血再灌注诱导的肝损伤的调节作用。
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Tumor necrosis factor-induced, superoxide-mediated neutrophil accumulation in cold ischemic/reperfused rat liver.肿瘤坏死因子诱导、超氧化物介导的中性粒细胞在冷缺血/再灌注大鼠肝脏中的积聚。
Hepatology. 1997 Jul;26(1):113-20. doi: 10.1053/jhep.1997.v26.pm0009214459.
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Mechanisms of reperfusion injury after warm ischemia of the liver.肝脏热缺血后再灌注损伤的机制
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Effect of Carolina rinse solution on Kupffer cell activation after reperfusion in rat liver transplantation.
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Factors in the pathophysiology of the liver ischemia-reperfusion injury.肝脏缺血再灌注损伤病理生理学中的因素。
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Role of microcirculation in hepatic ischemia/reperfusion injury.微循环在肝脏缺血/再灌注损伤中的作用。
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Acute ethanol binge followed by withdrawal regulates production of reactive oxygen species and cytokine-induced neutrophil chemoattractant and liver injury during reperfusion after hepatic ischemia.急性乙醇暴饮后戒断可调节肝缺血再灌注期间活性氧的产生、细胞因子诱导的中性粒细胞趋化因子及肝损伤。
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Activation of nuclear factor-kappaB during orthotopic liver transplantation in rats is protective and does not require Kupffer cells.大鼠原位肝移植过程中核因子-κB的激活具有保护作用，且不需要库普弗细胞。
Liver Transpl Surg. 1999 Jul;5(4):282-93. doi: 10.1002/lt.500050401.

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