Finardi G, Venco A, Saviotti M
G Ital Cardiol. 1976;6(3):483-95.
The authors studied the possible correlations between the behaviour of the systolic time intervals, as measured by the electrophonosphygmographic patterns, and the behaviour of the kinetocardiogram recorded on left precordium, in 23 cases of complete LBBB. The results tend to confirm the possibility of distinguishing the distal from the proximal level of the conduction defect in a non invasive way, as already indicated in literature. Evidence for the former is the observation of a lengthening of the isovolumetric contraction time, of an increase of the PEP/LVET and ICT/LVET ratios, of a pansystolic bulge in KCG with a pathological amplitude of the atrial waves, all showing a left ventricular dysfunction. Evidence for a proximal level of conduction defect is: the normal length of the ICT, the normal PEP/LVET and ICT/LVET ratios, the absence of pathological atrial waves in KCG. The presence of paradoxical outward movement (POM) in KCG also in the latter case, even though only rarely observable as a pansystolic bulge, could be related to the existence of a form of ventricular dyssynergy, due to the deranged conduction of the impulse (ventricular dyskinesis from inter- or intra-ventricular asynchrony).
作者研究了23例完全性左束支传导阻滞患者中，通过电音图模式测量的收缩期时间间期行为与左胸前记录的心动图行为之间可能存在的相关性。结果倾向于证实，如文献中已指出的那样，以非侵入性方式区分传导缺陷的远端和近端水平的可能性。支持前者的证据是观察到等容收缩时间延长、PEP/LVET和ICT/LVET比值增加、心动图中全收缩期隆起且心房波幅度异常，所有这些均显示左心室功能障碍。支持传导缺陷近端水平的证据是：ICT长度正常、PEP/LVET和ICT/LVET比值正常、心动图中无病理性心房波。在后一种情况下，即使很少观察到全收缩期隆起，但心动图中存在矛盾性向外运动（POM），这可能与一种心室协同失调形式的存在有关，这种协同失调是由于冲动传导紊乱（室间或室内不同步导致的心室运动障碍）。
