Raviele A, Piccolo E, Delise P, Caberlotto D, Di Pede F, Callegari E
G Ital Cardiol. 1981;11(8):1026-43.
Although in the last years very important knowledges have been obtained in the field of the intraventricular conduction disturbances, many electrocardiographic-vectorcardiographic findings concerning the left bundle branch block (LBBB) and their electrogenesis are still controversial. With the purpose to clarify this problem we have reviewed and analysed 23 cases in whom BCG-VCG patterns of "complete" or "incomplete" LBBB were induced by the premature right atrial stimulation (PRAS) during an electrophysiologic study in man. The analysis of these cases have demonstrated that: 1) the same LBBB pattern can be caused by a slowed conduction or block at different sites of left intraventricular conduction system i.e. not only in the main stem of the left bundle branch (LBB) (tronkular LBBB) but also within the bundle of His (intra-His LBBB) or in all the three fascicles of the LBB distally to its subdivision (divisional LBBB); 2) ECG-VCG are not able to distinguish the anatomical or functional site of slowed conduction or block; 3) the right or left axis deviation in the LBBB is not due to LBBB per se but it represents or a block at two different sites, i.e. a tronkular or intra His LBBB plus a block in the anterior or posterior subdivision of the LBB, or a block at only one side i.e. a tronkular, intra-His or divisional LBBB but with prevalent involvement of one fascicle of the LBB; 4) the ECG-VCG "incomplete" LBBB pattern is similar to that of the left ventricular enlargement; 5) figures of "atypical LBBB" are not specific of myocardial necrosis; 6) the criteria for the diagnosis of "complete" LBBB are not reliable. We conclude that, since LBBB pattern does not always correspond to a slowed conduction or block in the main stem of the LBB, the current terminology of LBBB is inappropriate and could be changed with another which considers the site of delayed activation and not the site of slowed conduction. Therefore we propose the following terminology: 1) generalized left ventricular activation delay instead of LBBB without axis deviation; 2) generalized left ventricular activation delay superiorly predominant instead of LBBB with left axis deviation; 3) generalized left ventricular activation delay inferiorly predominant instead of LBBB with right axis deviation.
尽管在过去几年里,在心室内传导障碍领域已经取得了非常重要的知识,但许多关于左束支传导阻滞(LBBB)及其电发生机制的心电图 - 向量心电图表现仍然存在争议。为了阐明这个问题,我们回顾并分析了23例在人体电生理研究中通过右心房早搏刺激(PRAS)诱发“完全性”或“不完全性”LBBB的BCG - VCG模式的病例。对这些病例的分析表明:1)相同的LBBB模式可由左心室内传导系统不同部位的传导减慢或阻滞引起,即不仅在左束支(LBB)的主干(主干型LBBB),也可在希氏束内(希氏束内LBBB)或在LBB分支远端的所有三个束支内(分支型LBBB);2)心电图 - 向量心电图无法区分传导减慢或阻滞的解剖或功能部位;3)LBBB中的右或左轴偏移并非由于LBBB本身,而是代表两个不同部位的阻滞,即主干型或希氏束内LBBB加上LBB前或后分支的阻滞,或仅一侧的阻滞,即主干型、希氏束内或分支型LBBB,但LBB的一个束支受累为主;4)心电图 - 向量心电图的“不完全性”LBBB模式与左心室扩大的模式相似;5)“非典型LBBB”图形并非心肌坏死所特有;6)“完全性”LBBB的诊断标准不可靠。我们得出结论,由于LBBB模式并不总是对应于LBB主干的传导减慢或阻滞,当前LBBB的术语不合适,可用另一种考虑延迟激动部位而非传导减慢部位的术语来替代。因此,我们提出以下术语:1)无轴偏移的LBBB用“广泛性左心室激动延迟”替代;2)左轴偏移的LBBB用“以向上为主的广泛性左心室激动延迟”替代;3)右轴偏移的LBBB用“以向下为主的广泛性左心室激动延迟”替代。