Mosonyi L, Juhász J, Surján L, Szilágyi G
Acta Med Acad Sci Hung. 1976;33(3):237-49.
Four cases with clinical and biochemical evidence of hyperparathyroidism are reported. The syndrome was due to DHT-intoxication of iatrogenic origin in the first case to histologically confirmed C-cell hyperplasia of the thyroid in the three others. The collective term "regulatory hyperparathyroidism" is porposed for the syndromes which, in distinction to secondary parathyroidisms, have a pathogenesis other than a negative calcium balance. Management of the syndrome is discussed with particular emphasis on the removal of the TCT-producing hyperplasia or adenoma, and the restoration of the electrolyte balance.
报告了4例有甲状旁腺功能亢进临床和生化证据的病例。第一例综合征是由医源性二氢睾酮中毒引起，其他三例是由组织学证实的甲状腺C细胞增生引起。对于与继发性甲状旁腺功能亢进不同，发病机制不是钙负平衡的综合征，提出了“调节性甲状旁腺功能亢进”这一统称。讨论了该综合征的处理方法，特别强调切除产生降钙素的增生或腺瘤以及恢复电解质平衡。
