Myocyte hypertrophy: the long and winding RhoA'd.
作者信息
Finkel T
机构信息
National Institutes of Health, Cardiology Branch, Bethesda, Maryland 20892-1650, USA.
出版信息
J Clin Invest. 1999 Jun;103(12):1619-20. doi: 10.1172/JCI7459.
Abstract
摘要
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Myocyte hypertrophy: the long and winding RhoA'd.心肌细胞肥大：漫长而曲折的RhoA之路。 （此翻译结合了一定意译，因为原句表述比较形象化，直接逐字翻译较难理解其确切含义，这种意译更符合语境表达的意思，供你参考。若你严格要求逐字翻译，可译为：心肌细胞肥大：漫长且曲折地受RhoA影响。 ）
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J Clin Invest. 1999 Jun;103(12):1627-34. doi: 10.1172/JCI6842.
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Rho family small G proteins play critical roles in mechanical stress-induced hypertrophic responses in cardiac myocytes.Rho家族小G蛋白在机械应力诱导的心肌细胞肥大反应中起关键作用。
Circ Res. 1999 Mar 5;84(4):458-66. doi: 10.1161/01.res.84.4.458.
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A requirement for the rac1 GTPase in the signal transduction pathway leading to cardiac myocyte hypertrophy.在导致心肌细胞肥大的信号转导途径中对rac1 GTP酶的需求。
J Clin Invest. 1998 Sep 1;102(5):929-37. doi: 10.1172/JCI2552.
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Proc Natl Acad Sci U S A. 1998 Aug 18;95(17):10140-5. doi: 10.1073/pnas.95.17.10140.
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Stimulation of the p38 mitogen-activated protein kinase pathway in neonatal rat ventricular myocytes by the G protein-coupled receptor agonists, endothelin-1 and phenylephrine: a role in cardiac myocyte hypertrophy?G蛋白偶联受体激动剂内皮素-1和去氧肾上腺素对新生大鼠心室肌细胞中p38丝裂原活化蛋白激酶途径的刺激作用：在心肌细胞肥大中起作用？
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