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The great escape: is immune evasion required for tumor progression?

作者信息

Villunger A, Strasser A

出版信息

Nat Med. 1999 Aug;5(8):874-5. doi: 10.1038/11311.

DOI:10.1038/11311
PMID:10426306
Abstract
摘要

相似文献

1
The great escape: is immune evasion required for tumor progression?大逃脱:肿瘤进展需要免疫逃逸吗?
Nat Med. 1999 Aug;5(8):874-5. doi: 10.1038/11311.
2
Escape from immune- and nonimmune-mediated tumor surveillance.逃脱免疫和非免疫介导的肿瘤监视。
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[The relationship between immunological defence and the development of tumours].[免疫防御与肿瘤发生发展之间的关系]
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4
MHC class I antigens, immune surveillance, and tumor immune escape.主要组织相容性复合体I类抗原、免疫监视与肿瘤免疫逃逸
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[Immune response and cancer].[免疫反应与癌症]
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Suppressive influences in the immune response to cancer.癌症免疫反应中的抑制性影响。
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Inhibition of T cell and natural killer cell function by adenosine and its contribution to immune evasion by tumor cells (Review).腺苷对T细胞和自然杀伤细胞功能的抑制及其对肿瘤细胞免疫逃逸的作用(综述)
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Tumor-host immune interactions and dendritic cell dysfunction.肿瘤-宿主免疫相互作用与树突状细胞功能障碍。
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Bee Venom Phospholipase A2, a Good "Chauffeur" for Delivering Tumor Antigen to the MHC I and MHC II Peptide-Loading Compartments of the Dendritic Cells: The Case of NY-ESO-1.
蜂毒磷脂酶A2,一种将肿瘤抗原递送至树突状细胞的MHC I和MHC II肽装载区室的优秀“司机”:以NY-ESO-1为例
PLoS One. 2013 Jun 18;8(6):e67645. doi: 10.1371/journal.pone.0067645. Print 2013.
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Alterations of HLA class I expression in human melanoma xenografts in immunodeficient mice occur frequently and are associated with higher tumorigenicity.免疫缺陷小鼠体内人黑色素瘤异种移植瘤中HLA I类分子表达的改变频繁发生,且与更高的致瘤性相关。
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Dendritic cells engineered to express CD40L continuously produce IL12 and resist negative signals from Tr1/Th3 dominated tumors.经基因工程改造以持续表达CD40L的树突状细胞可持续产生白细胞介素12,并抵抗来自Tr1/Th3主导型肿瘤的负性信号。
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Distribution of HLA class I altered phenotypes in colorectal carcinomas: high frequency of HLA haplotype loss associated with loss of heterozygosity in chromosome region 6p21.HLA I类改变表型在结直肠癌中的分布:HLA单倍型缺失的高频率与6号染色体区域6p21杂合性缺失相关。
Immunogenetics. 2004 Jul;56(4):244-53. doi: 10.1007/s00251-004-0692-z. Epub 2004 Jul 16.