Lahey B B, Waldman I D, McBurnett K
Department of Psychiatry, University of Chicago, IL 60637, USA.
J Child Psychol Psychiatry. 1999 Jul;40(5):669-82.
In this paper we have described an integrative causal model of the development of antisocial behavior in children and adolescents. The present model primarily integrates several previous models, but offers some new testable hypotheses. We believe that stable individual differences in propensity to antisocial behavior reflect variations in a number of dimensions of predisposing temperament and cognitive ability, each with its own genetic and environmental influences. The dimensions of predisposing temperament include oppositionality, harm avoidance, and callousness. Genetic influences are predicted to have only indirect effects on antisocial behavior via their influence on predisposition and on the youth's social environment. Environmental influences are expected to be important contributors to antisocial propensity, but these environmental influences reflect, in part, the genetic influences on the dimensions of predisposition (i.e. genotype-environment covariance). We also hypothesize that the levels of influence of the factors that determine individual differences in antisocial propensity change with development, such that genetic influences are of greater magnitude in early childhood and social influences contribute more strongly during later childhood and adolescence (both through independent effects and genotype-environment covariance). However, low levels of heritable predisposing child characteristics may protect against peer influences at all ages.
在本文中,我们描述了一个关于儿童和青少年反社会行为发展的综合因果模型。当前模型主要整合了几个先前的模型,但提出了一些新的可检验假设。我们认为,反社会行为倾向方面稳定的个体差异反映了多种易感性气质和认知能力维度的变化,每个维度都有其自身的遗传和环境影响。易感性气质维度包括对立性、回避伤害和冷酷无情。预计遗传影响仅通过其对易感性和青少年社会环境的影响,对反社会行为产生间接影响。环境影响预计是反社会倾向的重要促成因素,但这些环境影响部分反映了对易感性维度的遗传影响(即基因 - 环境协方差)。我们还假设,决定反社会倾向个体差异的因素的影响水平会随着发展而变化,使得遗传影响在幼儿期影响更大,而社会影响在童年后期和青少年期的作用更强(包括通过独立效应和基因 - 环境协方差)。然而,低水平的遗传性易感性儿童特征可能在所有年龄段都能抵御同伴影响。