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Nickel carcinogenesis, mutation, epigenetics, or selection.

作者信息

Costa M, Klein C B

出版信息

Environ Health Perspect. 1999 Sep;107(9):A438-9. doi: 10.1289/ehp.99107a438.

DOI:10.1289/ehp.99107a438
PMID:10464080
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1566440/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4d9e/1566440/e68659422f4d/envhper00514-0010-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4d9e/1566440/78776d53d38e/envhper00514-0010-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4d9e/1566440/e68659422f4d/envhper00514-0010-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4d9e/1566440/78776d53d38e/envhper00514-0010-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4d9e/1566440/e68659422f4d/envhper00514-0010-b.jpg

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1
Nickel carcinogenesis, mutation, epigenetics, or selection.镍的致癌作用、突变、表观遗传学或选择。
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Nickel carcinogenesis: epigenetics and hypoxia signaling.镍致癌作用:表观遗传学与缺氧信号传导
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本文引用的文献

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Nickel enhances telomeric silencing in Saccharomyces cerevisiae.
Mutat Res. 1999 Apr 6;440(2):121-30. doi: 10.1016/s1383-5718(99)00018-2.
2
Induction of activating transcription factor 1 by nickel and its role as a negative regulator of thrombospondin I gene expression.镍诱导激活转录因子1及其作为血小板反应蛋白I基因表达负调节因子的作用。
Cancer Res. 1997 Nov 15;57(22):5060-6.
3
DNA methylation, heterochromatin and epigenetic carcinogens.
Mutat Res. 1997 Apr;386(2):163-80. doi: 10.1016/s1383-5742(96)00052-x.
4
剖析致癌突变原的分子模态。
Environ Sci Pollut Res Int. 2022 Sep;29(41):62111-62159. doi: 10.1007/s11356-021-16726-w. Epub 2021 Oct 5.
4
Toxic effect of NiCl2 on development of the bursa of Fabricius in broiler chickens.氯化镍对肉鸡法氏囊发育的毒性作用。
Oncotarget. 2016 Jan 5;7(1):125-39. doi: 10.18632/oncotarget.6591.
5
Nickel chloride (NiCl2)-caused inflammatory responses via activation of NF-κB pathway and reduction of anti-inflammatory mediator expression in the kidney.氯化镍(NiCl₂)通过激活核因子κB(NF-κB)通路并降低肾脏中抗炎介质的表达引发炎症反应。
Oncotarget. 2015 Oct 6;6(30):28607-20. doi: 10.18632/oncotarget.5759.
6
Modulation of the PI3K/Akt Pathway and Bcl-2 Family Proteins Involved in Chicken's Tubular Apoptosis Induced by Nickel Chloride (NiCl₂).氯化镍(NiCl₂)诱导鸡肾小管细胞凋亡过程中PI3K/Akt信号通路及Bcl-2家族蛋白的调控作用
Int J Mol Sci. 2015 Sep 23;16(9):22989-3011. doi: 10.3390/ijms160922989.
7
The association between splenocyte apoptosis and alterations of Bax, Bcl-2 and caspase-3 mRNA expression, and oxidative stress induced by dietary nickel chloride in broilers.肉鸡脾脏细胞凋亡与Bax、Bcl-2和caspase-3 mRNA表达变化以及日粮氯化镍诱导的氧化应激之间的关系。
Int J Environ Res Public Health. 2013 Dec 17;10(12):7310-26. doi: 10.3390/ijerph10127310.
8
A panel study of occupational exposure to fine particulate matter and changes in DNA methylation over a single workday and years worked in boilermaker welders.一项针对锅炉工焊工在单个工作日及多年工作期间职业性接触细颗粒物与DNA甲基化变化的队列研究。
Environ Health. 2013 Jun 11;12(1):47. doi: 10.1186/1476-069X-12-47.
9
Spectroscopic and scanning electron microscopy studies of bioaccumulation of pollutants by algae.
Curr Microbiol. 2006 Aug;53(2):148-57. doi: 10.1007/s00284-005-0401-7. Epub 2006 Jun 26.
10
Essential role of PI-3K, ERKs and calcium signal pathways in nickel-induced VEGF expression.PI-3K、细胞外信号调节激酶(ERKs)和钙信号通路在镍诱导的血管内皮生长因子(VEGF)表达中的重要作用。
Mol Cell Biochem. 2005 Nov;279(1-2):35-43. doi: 10.1007/s11010-005-8214-3.
Loss of thrombospondin transcriptional activity in nickel-transformed cells.镍转化细胞中血小板反应蛋白转录活性的丧失。
Mol Cell Biol. 1994 Jan;14(1):851-8. doi: 10.1128/mcb.14.1.851-858.1994.
5
Model for the epigenetic mechanism of action of nongenotoxic carcinogens.
Am J Clin Nutr. 1995 Mar;61(3 Suppl):666S-669S. doi: 10.1093/ajcn/61.3.666S.
6
Mutagenic responses of nickel oxides and nickel sulfides in Chinese hamster V79 cell lines at the xanthine-guanine phosphoribosyl transferase locus.氧化镍和硫化镍在中国仓鼠V79细胞系黄嘌呤-鸟嘌呤磷酸核糖转移酶位点的诱变反应。
Mutat Res. 1993 Jun;300(1):63-72. doi: 10.1016/0165-1218(93)90141-y.
7
Carcinogenic nickel silences gene expression by chromatin condensation and DNA methylation: a new model for epigenetic carcinogens.致癌性镍通过染色质浓缩和DNA甲基化使基因表达沉默:一种表观遗传致癌物的新模型。
Mol Cell Biol. 1995 May;15(5):2547-57. doi: 10.1128/MCB.15.5.2547.
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Nonrandom chromosomal alterations in nickel-transformed Chinese hamster embryo cells.镍转化的中国仓鼠胚胎细胞中的非随机染色体改变。
Cancer Res. 1989 Nov 1;49(21):6032-8.
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Senescence of nickel-transformed cells by an X chromosome: possible epigenetic control.X染色体介导的镍转化细胞衰老:可能的表观遗传调控
Science. 1991 Feb 15;251(4995):796-9. doi: 10.1126/science.1990442.