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Unraveling hepatitis B virus infection of mice and men (and woodchucks and ducks).

作者信息

Menne S, Tennant B C

出版信息

Nat Med. 1999 Oct;5(10):1125-6. doi: 10.1038/13445.

DOI:10.1038/13445
PMID:10502809
Abstract
摘要

相似文献

1
Unraveling hepatitis B virus infection of mice and men (and woodchucks and ducks).解析小鼠、人类(以及土拨鼠和鸭子)的乙型肝炎病毒感染情况。
Nat Med. 1999 Oct;5(10):1125-6. doi: 10.1038/13445.
2
[T cell response to hepatitis B and C: from viral elimination to hepatocellular carcinoma].[乙肝和丙肝的T细胞反应:从病毒清除到肝细胞癌]
Dtsch Med Wochenschr. 2002 Oct 25;127(43):2277-9. doi: 10.1055/s-2002-35016.
3
Acute resolving woodchuck hepatitis virus (WHV) infection is associated with a strong cytotoxic T-lymphocyte response to a single WHV core peptide.急性消退性土拨鼠肝炎病毒(WHV)感染与针对单个WHV核心肽的强烈细胞毒性T淋巴细胞反应相关。
J Virol. 2007 Jul;81(13):7156-63. doi: 10.1128/JVI.02711-06. Epub 2007 Apr 25.
4
The pathogenesis of hepatocellular carcinoma is multifactorial event. Novel immunological treatment in prospect.肝细胞癌的发病机制是一个多因素事件。新型免疫治疗前景可期。
Clin Ter. 2004 May;155(5):187-99.
5
Chronic hepatitis B and hepatocarcinogenesis: does prevention of "collateral damage" bring the cure?
Hepatology. 2003 Mar;37(3):707-10. doi: 10.1002/hep.510370328.
6
Augmented hepatic interferon gamma expression and T-cell influx characterize acute hepatitis progressing to recovery and residual lifelong virus persistence in experimental adult woodchuck hepatitis virus infection.在实验性成年土拨鼠肝炎病毒感染中,肝脏中γ干扰素表达增强和T细胞流入是急性肝炎进展至恢复以及病毒持续终生残留的特征。
Hepatology. 2001 Nov;34(5):1049-59. doi: 10.1053/jhep.2001.29004.
7
Analysis of intrahepatic lymphocyte subsets in a transgenic mouse model of immune-mediated hepatocarcinogenesis.免疫介导的肝癌发生转基因小鼠模型中肝内淋巴细胞亚群的分析
J Exp Clin Cancer Res. 2006 Mar;25(1):55-7.
8
Hepatic iron overload induces hepatocellular carcinoma in transgenic mice expressing the hepatitis C virus polyprotein.肝铁过载在表达丙型肝炎病毒多聚蛋白的转基因小鼠中诱发肝细胞癌。
Gastroenterology. 2006 Jun;130(7):2087-98. doi: 10.1053/j.gastro.2006.02.060.
9
Comparative molecular pathogenesis of hepatocellular carcinomas.肝细胞癌的比较分子发病机制
Prog Clin Biol Res. 1992;376:173-85.
10
[The role of the virus specific T-cell response in acute and chronic HBV and HCV infection].[病毒特异性T细胞反应在急性和慢性乙肝病毒及丙肝病毒感染中的作用]
Z Gastroenterol. 2004 Jan;42(1):39-46. doi: 10.1055/s-2004-812688.

引用本文的文献

1
Down-regulation of hepatitis delta virus super-infection in the woodchuck model.下调土拨鼠模型中乙型肝炎病毒的超感染。
Virology. 2019 May;531:100-113. doi: 10.1016/j.virol.2019.03.002. Epub 2019 Mar 4.
2
Correlation of virus and host response markers with circulating immune complexes during acute and chronic woodchuck hepatitis virus infection.急性和慢性土拨鼠肝炎病毒感染期间病毒及宿主反应标志物与循环免疫复合物的相关性
J Virol. 2009 Feb;83(4):1579-91. doi: 10.1128/JVI.01934-08. Epub 2008 Dec 3.
3
Programmed death-1 expression is associated with the disease status in hepatitis B virus infection.
程序性死亡蛋白1的表达与乙型肝炎病毒感染中的疾病状态相关。
World J Gastroenterol. 2008 Jul 28;14(28):4551-7. doi: 10.3748/wjg.14.4551.
4
Molecular virology of hepatitis B virus for clinicians.临床医生的乙型肝炎病毒分子病毒学
Clin Liver Dis. 2007 Nov;11(4):685-706, vii. doi: 10.1016/j.cld.2007.08.002.
5
Antiviral effect of orally administered (-)-beta-D-2-aminopurine dioxolane in woodchucks with chronic woodchuck hepatitis virus infection.口服(-)-β-D-2-氨基嘌呤二氧戊环对慢性土拨鼠肝炎病毒感染土拨鼠的抗病毒作用。
Antimicrob Agents Chemother. 2007 Sep;51(9):3177-84. doi: 10.1128/AAC.00325-07. Epub 2007 Jul 2.
6
The woodchuck as an animal model for pathogenesis and therapy of chronic hepatitis B virus infection.土拨鼠作为慢性乙型肝炎病毒感染发病机制和治疗的动物模型。
World J Gastroenterol. 2007 Jan 7;13(1):104-24. doi: 10.3748/wjg.v13.i1.104.
7
Antiviral effect of oral administration of tenofovir disoproxil fumarate in woodchucks with chronic woodchuck hepatitis virus infection.口服富马酸替诺福韦二吡呋酯对慢性土拨鼠肝炎病毒感染土拨鼠的抗病毒作用。
Antimicrob Agents Chemother. 2005 Jul;49(7):2720-8. doi: 10.1128/AAC.49.7.2720-2728.2005.
8
Low doses of hepadnavirus induce infection of the lymphatic system that does not engage the liver.低剂量的嗜肝DNA病毒会引发不涉及肝脏的淋巴系统感染。
J Virol. 2004 Feb;78(4):1730-8. doi: 10.1128/jvi.78.4.1730-1738.2004.
9
Immunization with surface antigen vaccine alone and after treatment with 1-(2-fluoro-5-methyl-beta-L-arabinofuranosyl)-uracil (L-FMAU) breaks humoral and cell-mediated immune tolerance in chronic woodchuck hepatitis virus infection.单独使用表面抗原疫苗免疫以及在用1-(2-氟-5-甲基-β-L-阿拉伯呋喃糖基)-尿嘧啶(L-FMAU)治疗后进行免疫,可打破慢性土拨鼠肝炎病毒感染中的体液免疫和细胞介导的免疫耐受。
J Virol. 2002 Jun;76(11):5305-14. doi: 10.1128/jvi.76.11.5305-5314.2002.
10
Deficiencies in the acute-phase cell-mediated immune response to viral antigens are associated with development of chronic woodchuck hepatitis virus infection following neonatal inoculation.新生儿接种后,针对病毒抗原的急性期细胞介导免疫反应缺陷与慢性土拨鼠肝炎病毒感染的发生有关。
J Virol. 2002 Feb;76(4):1769-80. doi: 10.1128/jvi.76.4.1769-1780.2002.