Koga M, Minematsu K, Kazui S, Yasaka M, Yamaguchi T
Department of Medicine, National Cardiovascular Center, Osaka.
Rinsho Shinkeigaku. 1999 Jul;39(7):722-5.
Homolateral ataxia and crural paresis (HACP) is defined as predominantly crural paresis with ipsilateral ataxia, a variant of ataxic hemiparesis (AH), by Fisher and his colleagues. HACP usually resulted from lacunar infarction in the basis pontis at the junction of the upper one-third and inferior two-third of the pons, or in the posterior limb of the internal capsule. We reported a patient with HACP which was caused by an infarct in the paracentral gyrus irrigated by the anterior cerebral artery (ACA). He had had no cerebellar signs before the onset of HACP, although he had old small infarcts in the right pons, right thalamus and left cerebellar hemisphere. Neuroimaging and other clinical studies suggested that the mechanism of the present infarction was the most-likely embolic, but not lacunar. As far as we know, there has been only one abstract presentation of a patient with HACP due to ACA territory infarction in Japan, although five such cases were recently reported by Bogousslavsky and others.
同侧共济失调和小腿轻瘫（HACP）被费舍尔及其同事定义为以同侧共济失调为主的小腿轻瘫，是共济失调性偏瘫（AH）的一种变体。HACP通常由脑桥基底部上三分之一与下三分之二交界处或内囊后肢的腔隙性梗死引起。我们报告了一例由大脑前动脉（ACA）供血的中央旁回梗死导致HACP的患者。尽管他在右侧脑桥、右侧丘脑和左侧小脑半球有陈旧性小梗死灶，但在HACP发作前并无小脑体征。神经影像学和其他临床研究表明，此次梗死的机制很可能是栓塞性的，而非腔隙性的。据我们所知，在日本仅有一篇关于因ACA供血区梗死导致HACP患者的摘要报道，尽管最近博古斯拉夫斯基等人报告了5例此类病例。
