Interleukin-18, rheumatoid arthritis, and tissue destruction.
作者信息
Dayer J M
机构信息
Division of Immunology and Allergy, University Hospital, 1211 Geneva 14, Switzerland.
出版信息
J Clin Invest. 1999 Nov;104(10):1337-9. doi: 10.1172/JCI8731.
Abstract
摘要
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A proinflammatory role for IL-18 in rheumatoid arthritis.
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IL-18 is produced by articular chondrocytes and induces proinflammatory and catabolic responses.
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Cloning of a novel receptor subunit, AcPL, required for interleukin-18 signaling.
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Imbalance between interstitial collagenase and tissue inhibitor of metalloproteinases 1 in synoviocytes and fibroblasts upon direct contact with stimulated T lymphocytes: involvement of membrane-associated cytokines.
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