Parks S, Paul D W
Occupational and Environmental Medicine Division, University of Oklahoma Health Sciences Center-Oklahoma City, USA.
J Okla State Med Assoc. 2000 Feb;93(2):48-51.
A 45-year-old man working with ozone presents with evidence of sinusitis, mucus membrane irritation, sleep disturbance and shortness of breath. Naturally occurring or manmade, ozone may damage pulmonary alveolar type I cells at significant exposure levels. EPA and OSHA regulate exposure concentrations. Studies show dose responses with exposures. Supporting epidemiological studies are reviewed briefly. Limiting potential for excess exposure is key to prevention. Recognition of ozone as a potential exposure in the Oklahoma workplace is key to symptom management.
一名从事臭氧相关工作的45岁男性出现了鼻窦炎、黏膜刺激、睡眠障碍和呼吸急促的症状。臭氧无论是天然存在的还是人为制造的，在高暴露水平下都可能损害肺泡I型细胞。美国环境保护局（EPA）和职业安全与健康管理局（OSHA）对暴露浓度进行监管。研究表明暴露存在剂量反应关系。本文简要回顾了支持性的流行病学研究。限制过度暴露的可能性是预防的关键。认识到臭氧是俄克拉荷马州工作场所的潜在暴露源是症状管理的关键。
