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巨噬细胞通过凋亡依赖性吞噬作用清除甲型流感病毒感染的细胞以实现病毒清除。

Virus clearance through apoptosis-dependent phagocytosis of influenza A virus-infected cells by macrophages.

作者信息

Fujimoto I, Pan J, Takizawa T, Nakanishi Y

机构信息

Graduate School of Natural Science and Technology, Kanazawa University, Kanazawa, Ishikawa 920-0934, Japan.

出版信息

J Virol. 2000 Apr;74(7):3399-403. doi: 10.1128/jvi.74.7.3399-3403.2000.

Abstract

Some cultured cell lines undergo typical apoptosis upon infection with influenza virus. However, the release of replicated virus into the culture medium does not change when apoptosis is inhibited. Since apoptotic cells are heterophagically eliminated at early stages of the apoptosis pathway, we anticipated that the coexistence of phagocytic cells with virus-infected cells affects the extent of virus growth. When influenza A virus-infected HeLa cells were mixed with activated mouse peritoneal macrophages, efficient phagocytosis, which was abrogated in the presence of a caspase inhibitor, occurred. At the same time, the release of virus into the culture medium was completely inhibited, and this required direct contact between virus-infected cells and macrophages. Furthermore, an immunoelectron microscopic analysis detected influenza virus particles associated with phagosome-like structures within macrophages. These results indicate that apoptosis-dependent phagocytosis of virus-infected cells may lead to direct elimination of the pathogen.

摘要

一些培养的细胞系在感染流感病毒后会经历典型的凋亡。然而,当凋亡受到抑制时,复制病毒释放到培养基中的情况并未改变。由于凋亡细胞在凋亡途径的早期阶段会被异噬性清除,我们推测吞噬细胞与病毒感染细胞的共存会影响病毒生长的程度。当甲型流感病毒感染的HeLa细胞与活化的小鼠腹腔巨噬细胞混合时,发生了有效的吞噬作用,而这种作用在存在半胱天冬酶抑制剂时被消除。与此同时,病毒释放到培养基中的情况被完全抑制,这需要病毒感染细胞与巨噬细胞之间的直接接触。此外,免疫电子显微镜分析检测到巨噬细胞内与吞噬体样结构相关的流感病毒颗粒。这些结果表明,病毒感染细胞的凋亡依赖性吞噬作用可能导致病原体的直接清除。

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