RANKing the importance of measles virus in Paget's disease.
作者信息
Ross F P
机构信息
Department of Pathology, Washington University School of Medicine, Barnes-Jewish Hospital North, Mail Stop 90-31-649, 216 South Kingshighway, St. Louis, Missouri 63110, USA.
出版信息
J Clin Invest. 2000 Mar;105(5):555-8. doi: 10.1172/JCI9557.
Abstract
摘要
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本文引用的文献
1
Osteoclasts expressing the measles virus nucleocapsid gene display a pagetic phenotype.表达麻疹病毒核衣壳基因的破骨细胞呈现出畸形性骨炎表型。
J Clin Invest. 2000 Mar;105(5):607-14. doi: 10.1172/JCI8489.
2
The roles of osteoprotegerin and osteoprotegerin ligand in the paracrine regulation of bone resorption.骨保护素和骨保护素配体在骨吸收旁分泌调节中的作用。
J Bone Miner Res. 2000 Jan;15(1):2-12. doi: 10.1359/jbmr.2000.15.1.2.
3
Tumor necrosis factor alpha stimulates osteoclast differentiation by a mechanism independent of the ODF/RANKL-RANK interaction.肿瘤坏死因子α通过一种独立于骨桥蛋白/核因子κB受体活化因子配体-核因子κB受体活化因子相互作用的机制刺激破骨细胞分化。
J Exp Med. 2000 Jan 17;191(2):275-86. doi: 10.1084/jem.191.2.275.
4
TRANCE, a TNF family member, activates Akt/PKB through a signaling complex involving TRAF6 and c-Src.TRANCE是一种肿瘤坏死因子（TNF）家族成员，它通过一个涉及TRAF6和c-Src的信号复合物激活Akt/蛋白激酶B（PKB）。
Mol Cell. 1999 Dec;4(6):1041-9. doi: 10.1016/s1097-2765(00)80232-4.
5
Mutations in TNFRSF11A, affecting the signal peptide of RANK, cause familial expansile osteolysis.肿瘤坏死因子受体超家族成员11A（TNFRSF11A）的突变影响核因子κB受体激活因子（RANK）的信号肽，可导致家族性膨胀性骨质溶解。
Nat Genet. 2000 Jan;24(1):45-8. doi: 10.1038/71667.
6
Measles virus nucleocapsid transcript expression is not restricted to the osteoclast lineage in patients with Paget's disease of bone.在骨Paget病患者中，麻疹病毒核衣壳转录本表达并不局限于破骨细胞谱系。
Exp Hematol. 1999 Oct;27(10):1528-32. doi: 10.1016/s0301-472x(99)00097-1.
7
Update on the viral etiology of Paget's disease of bone.骨Paget病病毒病因学的最新进展。
J Bone Miner Res. 1999 Oct;14 Suppl 2:29-33. doi: 10.1002/jbmr.5650140207.
8
Cell biology of Paget's disease.佩吉特病的细胞生物学
J Bone Miner Res. 1999 Oct;14 Suppl 2:3-8. doi: 10.1002/jbmr.5650140203.
9
NF-kappaB activation by tumour necrosis factor requires the Akt serine-threonine kinase.肿瘤坏死因子激活核因子-κB需要Akt丝氨酸-苏氨酸激酶。
Nature. 1999 Sep 2;401(6748):82-5. doi: 10.1038/43466.
10
Targeted disruption of Traf5 gene causes defects in CD40- and CD27-mediated lymphocyte activation.Traf5基因的靶向破坏导致CD40和CD27介导的淋巴细胞活化缺陷。
Proc Natl Acad Sci U S A. 1999 Aug 17;96(17):9803-8. doi: 10.1073/pnas.96.17.9803.
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