Krylov V N, Yankovsky N K
J Virol. 1975 Jan;15(1):22-6. doi: 10.1128/JVI.15.1.22-26.1975.
Mutations of bacteriophage T4B were found which suppress the lysis defect of both gene stII mutants and gene e mutants. The suppressor mutations belong to a new gene, stIII, of phage T4B. Gene stIII is located on the genetic map of T4B between genes stI and e. stIII mutants sometimes form star plaques on Escherichia coli B. The latent period on E. coli 594, but not E. coli B, is shorter with stIII mutants than that with wild-type phage. The premature lysis of E. coli 594 infected with stIII phage does not depend on the expression of both stII+ and e+ function. StIII allele is dominant over the stIII+ with respect to both the ability to suppress the stII defect and the early lysis of infected E. coli 594 cultures.
发现噬菌体T4B的突变可抑制基因stII突变体和基因e突变体的裂解缺陷。这些抑制突变属于噬菌体T4B的一个新基因stIII。基因stIII位于T4B的遗传图谱上，在基因stI和e之间。stIII突变体有时在大肠杆菌B上形成星状噬菌斑。与野生型噬菌体相比，stIII突变体在大肠杆菌594上的潜伏期较短，但在大肠杆菌B上并非如此。感染stIII噬菌体的大肠杆菌594的过早裂解不依赖于stII+和e+功能的表达。就抑制stII缺陷的能力和感染的大肠杆菌594培养物的早期裂解而言，StIII等位基因相对于stIII+是显性的。
