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维拉帕米和阿替洛尔引发的心源性休克:静脉补钙治疗经验的病例报告

Cardiogenic shock triggered by verapamil and atenolol: a case report of therapeutic experience with intravenous calcium.

作者信息

Sakurai H, Kei M, Matsubara K, Yokouchi K, Hattori K, Ichihashi R, Hirakawa Y, Tsukamoto H, Saburi Y

机构信息

Department of Cardiology, Toki General Hospital, Toki City, Gifu, Japan.

出版信息

Jpn Circ J. 2000 Nov;64(11):893-6. doi: 10.1253/jcj.64.893.

DOI:10.1253/jcj.64.893
PMID:11110438
Abstract

Cardiogenic shock developed in a 72-year-old Japanese woman during combination therapy with verapamil and atenolol for recurrent supraventricular arrhythmia. She had coronary atherosclerosis, liver cirrhosis and bradycardia-tachycardia syndrome. Despite of the high-dose catecholamines and counterpulsation, she progressively deteriorated. Bolus administration of intravenous calcium chloride (CaCl2) immediately resolved her hemodynamic collapse.

摘要

一名72岁日本女性在使用维拉帕米和阿替洛尔联合治疗复发性室上性心律失常期间发生心源性休克。她患有冠状动脉粥样硬化、肝硬化和心动过缓-心动过速综合征。尽管使用了大剂量儿茶酚胺和反搏治疗,她的病情仍逐渐恶化。静脉推注氯化钙(CaCl2)立即缓解了她的血流动力学崩溃。

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