肿瘤抑制的单倍剂量不足：单身且长寿的风险。 - Suppr

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超能文献

Haploinsufficiency for tumor suppression: the hazards of being single and living a long time.

作者信息

Largaespada D A

机构信息

Department of Genetics, Cell Biology, and Development, University of Minnesota, Minneapolis, Minnesota 55455, USA.

出版信息

J Exp Med. 2001 Feb 19;193(4):F15-8. doi: 10.1084/jem.193.4.f15.

DOI:10.1084/jem.193.4.f15

PMID:11181707

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2195912/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/5854/2195912/66ee918ff351/JEM010122.f1.jpg

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本文引用的文献

J Exp Med. 2001 Feb 19;193(4):531-43. doi: 10.1084/jem.193.4.531.

Role of promyelocytic leukemia (PML) protein in tumor suppression.早幼粒细胞白血病（PML）蛋白在肿瘤抑制中的作用。

J Exp Med. 2001 Feb 19;193(4):521-29. doi: 10.1084/jem.193.4.521.

Acute promyelocytic leukemia (AML-M3)--Part 2: Molecular defect, DNA diagnosis, and proposed models of leukemogenesis and differentiation therapy.急性早幼粒细胞白血病（AML-M3）——第二部分：分子缺陷、DNA诊断以及白血病发生和分化治疗的提出模型

Clin Lab Sci. 2000 Spring;13(2):106-16.

Characterization of acute promyelocytic leukemia cases lacking the classic t(15;17): results of the European Working Party. Groupe Français de Cytogénétique Hématologique, Groupe de Français d'Hematologie Cellulaire, UK Cancer Cytogenetics Group and BIOMED 1 European Community-Concerted Action "Molecular Cytogenetic Diagnosis in Haematological Malignancies".缺乏经典t(15;17)的急性早幼粒细胞白血病病例的特征：欧洲工作组的结果。法国血液细胞遗传学小组、法国血液细胞学小组、英国癌症细胞遗传学小组以及生物医药1欧洲共同体协调行动“血液系统恶性肿瘤的分子细胞遗传学诊断”

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