Stice E
Department of Psychology, 330 Mezes Hall, University of Texas at Austin, Austin, Texas 78712, USA.
J Abnorm Psychol. 2001 Feb;110(1):124-35. doi: 10.1037//0021-843x.110.1.124.
Because there have been few longitudinal investigations of integrative etiological theories of bulimia nervosa, this study prospectively tested the dual-pathway model using random regression growth curve models and data from a 3-wave community sample of adolescent girls (N = 231). Initial pressure to be thin and thin-ideal internalization predicted subsequent growth in body dissatisfaction, initial body dissatisfaction predicted growth in dieting and negative affect, and initial dieting and negative affect predicted growth in bulimic symptoms. There was prospective evidence for most of the hypothesized mediational effects. Results are consistent with the assertion that pressure to be thin, thin-ideal internalization, body dissatisfaction, dieting, and negative affect are risk factors for bulimic pathology and provide support for the dual-pathway model.
由于对神经性贪食症综合病因理论的纵向研究较少，本研究使用随机回归增长曲线模型和来自青少年女孩三波社区样本（N = 231）的数据，对双路径模型进行了前瞻性测试。最初的瘦身压力和瘦身理想内化预测了随后身体不满情绪的增长，最初的身体不满情绪预测了节食和负面影响的增长，最初的节食和负面影响预测了贪食症状的增长。对于大多数假设的中介效应，都有前瞻性证据。结果与以下观点一致，即瘦身压力、瘦身理想内化、身体不满、节食和负面影响是贪食症病理的风险因素，并为双路径模型提供了支持。
