Stice E
Department of Psychiatry, Stanford University, California 94305-5722, USA.
Int J Eat Disord. 1998 Apr;23(3):243-60. doi: 10.1002/(sici)1098-108x(199804)23:3<243::aid-eat2>3.0.co;2-j.
Although theorists have proposed that restraint and negative affect cause bulimia nervosa, it is possible that bulimic pathology promotes restraint and negative affect, or that bulimic symptoms are reciprocally related to these two factors. The present study tested these competing models.
Longitudinal data from a community sample of adolescent females (N = 218) was used to test these alternative models.
Prospective correlations suggested that bulimic pathology was reciprocally related to both restraint and negative affect. However, in more stringent tests controlling for the temporal stability of these factors, restraint was not related to subsequent bulimic symptoms, but bulimic pathology predicted future restraint. Negative affect and bulimic symptoms were not related over time when the stability of these factors was controlled, but they did show contemporaneous reciprocal relations.
Results provide some support for the negative affect model of bulimia, but raise questions about the restraint model.
尽管理论学家提出克制和消极情绪会导致神经性贪食症，但也有可能是贪食症病理促使了克制和消极情绪的产生，或者贪食症状与这两个因素相互关联。本研究对这些相互竞争的模型进行了检验。
采用来自青少年女性社区样本（N = 218）的纵向数据来检验这些替代模型。
前瞻性相关性表明，贪食症病理与克制和消极情绪均相互关联。然而，在对这些因素的时间稳定性进行控制的更严格检验中，克制与随后的贪食症状无关，但贪食症病理可预测未来的克制。当控制这些因素的稳定性时，消极情绪与贪食症状随时间并无关联，但它们确实呈现出同期的相互关系。
研究结果为神经性贪食症的消极情绪模型提供了一些支持，但对克制模型提出了疑问。
