• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Simultaneous occurrence of bcr-abl and inv16 in a case of M1 acute myeloid leukemia.

作者信息

Svaldi M, Lanthaler A, Venturi R, Coser P, Mitterer M

出版信息

Leukemia. 2001 Apr;15(4):695. doi: 10.1038/sj.leu.2402082.

DOI:10.1038/sj.leu.2402082
PMID:11368385
Abstract
摘要

相似文献

1
Simultaneous occurrence of bcr-abl and inv16 in a case of M1 acute myeloid leukemia.
Leukemia. 2001 Apr;15(4):695. doi: 10.1038/sj.leu.2402082.
2
Coexistence of inversion 16 and the Philadelphia chromosome in acute and chronic myeloid leukemias : report of six cases and review of literature.急性和慢性髓系白血病中16号染色体倒位与费城染色体共存：6例报告及文献复习
Am J Clin Pathol. 2006 Feb;125(2):260-6. doi: 10.1309/F0MX-5CL8-CEDY-3W86.
3
De novo acute myeloid leukemia with Philadelphia chromosome (BCR-ABL) and inversion 16 (CBFB-MYH11): report of two cases and review of the literature.伴有费城染色体（BCR-ABL）和16号染色体倒位（CBFB-MYH11）的新发急性髓系白血病：两例报告并文献复习
Leuk Lymphoma. 2011 Mar;52(3):531-5. doi: 10.3109/10428194.2010.538941. Epub 2011 Feb 1.
4
The NDE1 gene is disrupted by the inv(16) in 90% of cases with CBFB-MYH11-positive acute myeloid leukemia.在90%的CBFB-MYH11阳性急性髓系白血病病例中，NDE1基因被inv(16)破坏。
Leukemia. 2010 Apr;24(4):857-9. doi: 10.1038/leu.2009.290. Epub 2010 Jan 14.
5
Identification of a novel CBFB-MYH11 fusion transcript in a patient with AML and inversion of chromosome 16.
Leukemia. 1998 Dec;12(12):2034-5. doi: 10.1038/sj.leu.2401209.
6
[Research on the molecular etiology of inversion of chromosome 16 in the patients with acute myeloid leukemia].[急性髓系白血病患者16号染色体倒位的分子病因学研究]
Zhonghua Xue Ye Xue Za Zhi. 1998 Jan;19(1):49-51.
7
Deletion of 3'CBFbeta in an inv(16)(p13.lq22) ascertained by fluorescence in situ hybridization and reverse-transcriptase polymerase chain reaction.通过荧光原位杂交和逆转录聚合酶链反应确定在inv(16)(p13.lq22)中3'CBFβ的缺失。
Cancer Genet Cytogenet. 2007 Jan 1;172(1):92-4. doi: 10.1016/j.cancergencyto.2006.08.006.
8
Deletion of chromosome 22 without bcr rearrangement and without juxtaposition of c-abl in a case of acute myeloid leukemia.1例急性髓系白血病患者中22号染色体缺失，无bcr重排且c-abl无并置现象。
Cancer Genet Cytogenet. 1993 Jun;67(2):141-4. doi: 10.1016/0165-4608(93)90168-l.
9
Early transformation to acute myeloblastic leukaemia with the acquisition of inv(16) in Ph positive chronic granulocytic leukaemia.伴有inv(16)获得的Ph阳性慢性粒细胞白血病早期转化为急性髓细胞白血病
Leuk Res. 1997 May;21(5):473-4. doi: 10.1016/s0145-2126(97)00070-2.
10
A BCR-JAK2 fusion gene as the result of a t(9;22)(p24;q11) in a patient with acute myeloid leukemia.一名急性髓系白血病患者因t(9;22)(p24;q11)产生的一种BCR-JAK2融合基因。
Cancer Genet Cytogenet. 2008 Jun;183(2):105-8. doi: 10.1016/j.cancergencyto.2008.02.005.

引用本文的文献

1
Concurrent and core binding factor beta rearrangement in de novo acute myeloid leukemia: A case report and review of literature.初发急性髓系白血病中的并发及核心结合因子β重排：一例病例报告及文献综述
EJHaem. 2024 Apr 17;5(3):607-615. doi: 10.1002/jha2.895. eCollection 2024 Jun.
2
A novel subclonal rearrangement of the STRN3::PDGFRB gene in de novo acute myeloid leukemia with NPM1 mutation and its leukemogenic effects.伴有NPM1突变的初发急性髓系白血病中STRN3::PDGFRB基因的一种新型亚克隆重排及其致白血病作用。
Cancer Gene Ther. 2023 Nov;30(11):1471-1484. doi: 10.1038/s41417-023-00651-w. Epub 2023 Aug 7.
3
De Novo Acute Myeloid Leukemia with Combined and Gene Rearrangements: A Case Report and Review of Literature.
伴有复合及基因重排的新发急性髓系白血病：一例报告并文献复习
Case Rep Hematol. 2020 Dec 16;2020:8822670. doi: 10.1155/2020/8822670. eCollection 2020.
4
[Hematologic malignancies with coexisting t(9;22) and inv(16) chromosomal abnormalities: report of three cases and literature review].[合并t(9;22)和inv(16)染色体异常的血液系统恶性肿瘤：三例报告及文献复习]
Zhonghua Xue Ye Xue Za Zhi. 2020 Nov 14;41(11):937-940. doi: 10.3760/cma.j.issn.0253-2727.2020.11.010.
5
Coexistence of p210 and CBFβ-MYH11 fusion genes in myeloid leukemia: A report of 4 cases.髓系白血病中p210和CBFβ-MYH11融合基因的共存：4例报告
Oncol Lett. 2017 Nov;14(5):5171-5178. doi: 10.3892/ol.2017.6812. Epub 2017 Aug 24.
6
t(9;22) as secondary alteration in core-binding factor de novo acute myeloid leukemia.t(9;22)作为核心结合因子原发性急性髓系白血病的继发性改变。
Am J Hematol. 2015 Nov;90(11):E211-2. doi: 10.1002/ajh.24143. Epub 2015 Oct 12.
7
Characteristics of hematologic malignancies with coexisting t(9;22) and inv(16) chromosomal abnormalities.同时存在t(9;22)和inv(16)染色体异常的血液系统恶性肿瘤的特征
Blood Res. 2014 Mar;49(1):22-8. doi: 10.5045/br.2014.49.1.22. Epub 2014 Mar 24.
8
p210 BCR/ABL1 as a secondary change in a patient with acute myelomonocytic leukemia (M4Eo) with inv(16).伴有 inv(16)的急性髓单核细胞白血病（M4Eo）患者中出现的继发改变的 BCR/ABL1
Int J Hematol. 2012 Dec;96(6):814-7. doi: 10.1007/s12185-012-1190-y. Epub 2012 Oct 11.
9
Patterns of BCR breakpoints in patients with coexisting inv(16)(p13.1q22) and t(9;22)(q34;q11.2).同时存在inv(16)(p13.1q22)和t(9;22)(q34;q11.2)的患者中BCR断点模式
Int J Hematol. 2012 Mar;95(3):324-6. doi: 10.1007/s12185-011-0990-9. Epub 2012 Feb 25.