Aĭrapetian S N
Biofizika. 1975 May-Jun;20(3):462-6.
Depolarization current decreases and hyperpolarization current increases the amplitude of tracing hyperpolarization of the neuron action potential. Calcium-defficient solution supresses the tracing depolarization, and turns the rhythmical activity of the neuron into the flashlike one. An increase of outer concentration of potassium ions decreases the tracing depolarization. The latter is suppressed completely when the membrane behaves as a potassium electrode. The suppressing effect of the increase of potassium outer concentration on tracing hyperpolarization decreases with a decrease of calcium ions content in the medium. When an active release of sodium ions from the cell is inhibited with DNP and substitution of sodium ions by lithium ions the tracing hyperpolarization of the action potential is suppressed. The tracing hyperpolarization is also suppressed during the shunting of the electrogenic effect of potassium pump with the outcoming current of chlorine ions. It is suggested that the tracing hyperpolarization of the single action potential is due to the calcium-dependent fraction of electrogenic release of sodium ions from the cell.
去极化电流减小，而超极化电流增加神经元动作电位超极化的描记幅度。缺钙溶液抑制描记去极化，并将神经元的节律性活动转变为闪光样活动。细胞外钾离子浓度增加会降低描记去极化。当膜表现为钾电极时，后者被完全抑制。细胞外钾离子浓度增加对描记超极化的抑制作用随培养基中钙离子含量的降低而减小。当用二硝基酚抑制细胞中钠离子的主动释放并用锂离子替代钠离子时，动作电位的描记超极化被抑制。在用氯离子外流分流钾泵的生电效应期间，描记超极化也被抑制。有人提出，单个动作电位的描记超极化是由于细胞中钠离子生电释放的钙依赖性部分所致。
