Aung T, Looi A L, Chew P T
Singapore National Eye Centre, Tan Tock Seng Hospital, Singapore, National University of Singapore.
Acta Ophthalmol Scand. 2001 Jun;79(3):298-300. doi: 10.1034/j.1600-0420.2001.790318.x.
To determine the frequency and type of visual field loss six months after an episode of acute primary angle closure (APAC), and to identify risk factors for the development of such field loss.
This was a cross sectional observational study. All patients who suffered from an episode of APAC at two Singapore hospitals over a one-year period underwent static automated threshold perimetry six months after presentation.
38% of (29) patients with APAC have significant visual field defects six months after the acute episode. The majority of those with abnormal fields had hemifield defects, consistent with nerve fiber bundle pattern loss. Those with pre-existing chronic glaucoma or who develop a rise in intraocular pressure during follow-up are at risk of visual field loss. The risk of visual field loss is also significant if the duration of symptoms exceeds 7 days.
The frequency of visual field loss at 6 months after APAC was low at only 38%. As the majority of eyes have no evidence of detectable functional damage developing as a consequence of the acute episode, APAC may not be blinding if treated promptly and adequately.
确定急性原发性闭角型青光眼（APAC）发作6个月后视野缺损的频率和类型，并识别发生此类视野缺损的危险因素。
这是一项横断面观察性研究。在一年时间里，两家新加坡医院所有发生过APAC发作的患者在就诊6个月后均接受了静态自动阈值视野检查。
38%（29例）的APAC患者在急性发作6个月后出现明显的视野缺损。大多数视野异常的患者存在半侧视野缺损，符合神经纤维束型缺损。既往患有慢性青光眼或随访期间眼压升高的患者有视野缺损的风险。如果症状持续时间超过7天，视野缺损的风险也很高。
APAC发作6个月后视野缺损的频率较低，仅为38%。由于大多数眼睛没有因急性发作而出现可检测到的功能性损害的证据，因此如果及时、充分地治疗，APAC可能不会导致失明。
