Arvin A M
G-312, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, CA 94305, USA.
Curr Opin Microbiol. 2001 Aug;4(4):442-9. doi: 10.1016/s1369-5274(00)00233-2.
Cosmid-based mutagenesis and methods to examine varicella-zoster virus (VZV) tropism for differentiated human cells in vivo provide new information about molecular mechanisms of VZV infection. How specific VZV gene products contribute to viral replication has been further defined, and effects of VZV on expression of cellular genes have been demonstrated.
基于黏粒的诱变技术以及在体内检测水痘带状疱疹病毒（VZV）对分化人细胞的嗜性的方法，为VZV感染的分子机制提供了新信息。特定VZV基因产物如何促进病毒复制已得到进一步明确，并且VZV对细胞基因表达的影响也已得到证实。
