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移植物抗宿主病和移植物抗白血病反应中的细胞因子。

Cytokines in graft-versus-host disease and the graft-versus-leukemia reaction.

作者信息

Deeg H J

机构信息

Fred Hutchinson Cancer Research Center and University of Washington, Seattle 98109-1024, USA.

出版信息

Int J Hematol. 2001 Jul;74(1):26-32. doi: 10.1007/BF02982546.

Abstract

Graft-versus-host disease (GVHD) is the major complication after allogeneic hemopoietic stem cell transplantation. GVHD is destructive by itself and sets the stage for other sequelae, in particular, overwhelming infections. Recent investigations have improved our understanding of the underlying pathophysiology of GVHD. There are now compelling data on the role of host tissue destruction as the initial insult, extensive interactions of cellular donor and host components, a complex network of cytokines, adhesion molecules, and other components in the development of GVHD. The improved understanding of interactions among various signals is likely to allow for the development of new prophylactic strategies. A review of the data shows, however, that results are very dependent upon the models used. It is difficult or impossible to separate completely the discussion of cytokines that affect hemopoietic cells from discussion of cytokines that exert effects on immune cells. Furthermore, secondary effects on immune cells via hemopoietic cells complicate the picture. Application of the principles of cytokine signaling to the clinical setting may necessitate new trial design structures that take into consideration donor and host characteristics as well as the kinetics of GVHD development.

摘要

移植物抗宿主病(GVHD)是异基因造血干细胞移植后的主要并发症。GVHD本身具有破坏性,并为其他后遗症奠定了基础,尤其是严重感染。最近的研究增进了我们对GVHD潜在病理生理学的理解。目前有令人信服的数据表明,宿主组织破坏作为初始损伤的作用、细胞供体和宿主成分的广泛相互作用、细胞因子、黏附分子及其他成分在GVHD发生发展中的复杂网络。对各种信号间相互作用的更好理解可能会促成新的预防策略的开发。然而,对数据的回顾表明,结果非常依赖于所使用的模型。很难或不可能将影响造血细胞的细胞因子讨论与对免疫细胞起作用的细胞因子讨论完全分开。此外,通过造血细胞对免疫细胞的继发性影响使情况变得复杂。将细胞因子信号传导原理应用于临床可能需要新的试验设计结构,该结构要考虑供体和宿主特征以及GVHD发生发展的动力学。

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