So what is critically lacking with coronary atherosclerotic plaques? Perhaps the antithrombotic control.
作者信息
Edgington T S
机构信息
Departments of Vascular Biology and Immunology, The Scripps Research Institute, La Jolla, California 92037, USA.
出版信息
Am J Pathol. 2001 Sep;159(3):795-6. doi: 10.1016/S0002-9440(10)61752-X.
Abstract
摘要
相似文献
1
So what is critically lacking with coronary atherosclerotic plaques? Perhaps the antithrombotic control.
Am J Pathol. 2001 Sep;159(3):795-6. doi: 10.1016/S0002-9440(10)61752-X.
2
Presence of thrombosis in coronary atherosclerotic plaques.
J Cardiovasc Risk. 1995 Jun;2(3):189-96.
3
[From atherosclerotic plaque to coronary thrombus: pathophysiologic aspects of acute coronary syndromes].
G Ital Cardiol. 1998 May;28(5):586-95.
4
Present concepts of coronary atherosclerosis-thrombosis, therapeutic implications and perspectives.冠状动脉粥样硬化-血栓形成的当前概念、治疗意义及前景。
Arch Mal Coeur Vaiss. 1997 Nov;90 Spec No 6:41-7.
5
Atherosclerosis and plaque rupture: an update.
J Pak Med Assoc. 1999 Feb;49(2):37-43.
6
Factors determining and prevention of atherosclerotic plaque rupture.动脉粥样硬化斑块破裂的决定因素及预防
Postgrad Med J. 1994;70 Suppl 1:S43-5.
7
Association of coronary plaque rupture and atherosclerotic inflammation.冠状动脉斑块破裂与动脉粥样硬化炎症的关联。
J Pathol. 1997 Jan;181(1):93-9. doi: 10.1002/(SICI)1096-9896(199701)181:1<93::AID-PATH696>3.0.CO;2-H.
8
The way ahead in the management of unstable coronary artery disease.
Am J Cardiol. 1997 Sep 4;80(5A):64E-67E. doi: 10.1016/s0002-9149(97)00494-3.
9
Significance of multiple coronary artery thrombi. A consequence of diffuse atherosclerotic disease?多发性冠状动脉血栓的意义。弥漫性动脉粥样硬化疾病的一个后果？
Ital Heart J. 2000 Dec;1(12):832-4.
10
The composition of coronary-artery plaques.冠状动脉斑块的组成。
N Engl J Med. 1997 May 1;336(18):1312-4. doi: 10.1056/NEJM199705013361809.
本文引用的文献
1
Down-regulation of endothelial expression of endothelial cell protein C receptor and thrombomodulin in coronary atherosclerosis.冠状动脉粥样硬化中内皮细胞蛋白C受体和血栓调节蛋白在内皮表达的下调。
Am J Pathol. 2001 Sep;159(3):797-802. doi: 10.1016/S0002-9440(10)61753-1.
2
Coronary artery injury and the biology of atherosclerosis: inflammation, thrombosis, and stabilization.
Am J Cardiol. 2000 Oct 19;86(8B):3J-8J; discussion 8J-9J. doi: 10.1016/s0002-9149(00)01339-4.
3
Thrombin signalling and protease-activated receptors.凝血酶信号传导与蛋白酶激活受体
Nature. 2000 Sep 14;407(6801):258-64. doi: 10.1038/35025229.
4
Lessons from sudden coronary death: a comprehensive morphological classification scheme for atherosclerotic lesions.心源性猝死的经验教训：动脉粥样硬化病变的综合形态学分类方案
Arterioscler Thromb Vasc Biol. 2000 May;20(5):1262-75. doi: 10.1161/01.atv.20.5.1262.
5
Regulation of blood coagulation.血液凝固的调节
Biochim Biophys Acta. 2000 Mar 7;1477(1-2):349-60. doi: 10.1016/s0167-4838(99)00266-6.
6
The structural basis of function of the TF. VIIa complex in the cellular initiation of coagulation.凝血因子VIIa复合物在细胞内凝血起始过程中发挥功能的结构基础。
Thromb Haemost. 1997 Jul;78(1):401-5.
7
Localization of tissue factor in the normal vessel wall and in the atherosclerotic plaque.组织因子在正常血管壁和动脉粥样硬化斑块中的定位。
Proc Natl Acad Sci U S A. 1989 Apr;86(8):2839-43. doi: 10.1073/pnas.86.8.2839.
Zotero 插件