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细胞因子信号转导抑制蛋白(SOCS):细胞因子信号传导的负调节因子。

SOCS proteins: negative regulators of cytokine signaling.

作者信息

Krebs D L, Hilton D J

机构信息

The Walter and Eliza Hall Institute of Medical Research and the Cooperative Research Center for Cellular Growth Factors, Royal Melbourne Hospital, Victoria, Australia.

出版信息

Stem Cells. 2001;19(5):378-87. doi: 10.1634/stemcells.19-5-378.

Abstract

Cytokines regulate the growth and differentiation of cells by binding to cell-surface receptors and activating intracellular signal transduction cascades such as the JAK-STAT pathway. Cytokine signaling is negatively regulated with respect to both magnitude and duration, and it is now clear that the suppressor of cytokine signaling (SOCS) family of proteins (SOCS1-SOCS7 and CIS) contributes significantly to this process. Transcripts encoding CIS, SOCS1, SOCS2, and SOCS3 are upregulated in response to cytokine stimulation, and the corresponding SOCS proteins inhibit cytokine-induced signaling pathways. SOCS proteins therefore form part of a classical negative feedback circuit. SOCS family members modulate signaling by several mechanisms, which include inactivation of the Janus kinases (JAKs), blocking access of the signal transducers and activators of transcription (STATs) to receptor binding sites, and ubiquitination of signaling proteins and their subsequent targeting to the proteasome. Gene targeting has been used to generate mice lacking socs1, socs2, or socs3, in order to elucidate the physiological function of these SOCS family members. The analysis of socs1(-/-) mice has revealed that SOCS1 plays a key role in the negative regulation of interferon-gamma signaling and in T cell differentiation. Socs2(-/-) mice are 30%-40% larger than wild-type mice, demonstrating that SOCS2 is a critical regulator of postnatal growth. Additionally, the study of embryos lacking socs3 has revealed that SOCS3 is an important regulator of fetal liver hematopoiesis. The biological role of other SOCS proteins remains to be determined.

摘要

细胞因子通过与细胞表面受体结合并激活细胞内信号转导级联反应(如JAK-STAT途径)来调节细胞的生长和分化。细胞因子信号传导在强度和持续时间方面均受到负调控,现在很清楚,细胞因子信号传导抑制因子(SOCS)蛋白家族(SOCS1-SOCS7和CIS)在这一过程中发挥着重要作用。编码CIS、SOCS1、SOCS2和SOCS3的转录本在细胞因子刺激下会上调,相应的SOCS蛋白会抑制细胞因子诱导的信号通路。因此,SOCS蛋白构成了经典负反馈回路的一部分。SOCS家族成员通过多种机制调节信号传导,这些机制包括使Janus激酶(JAKs)失活、阻止信号转导子和转录激活子(STATs)进入受体结合位点,以及对信号蛋白进行泛素化并随后将其靶向蛋白酶体。基因靶向已被用于培育缺乏socs1、socs2或socs3的小鼠,以阐明这些SOCS家族成员的生理功能。对socs1(-/-)小鼠的分析表明,SOCS1在干扰素-γ信号传导的负调控和T细胞分化中起关键作用。Socs2(-/-)小鼠比野生型小鼠大30%-40%,表明SOCS2是出生后生长的关键调节因子。此外,对缺乏socs3的胚胎的研究表明,SOCS3是胎儿肝脏造血的重要调节因子。其他SOCS蛋白的生物学作用仍有待确定。

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