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起源于冠状窦肌组织的反复性房性心动过速。

Iterative atrial tachycardia originating from the coronary sinus musculature.

作者信息

Tritto M, Zardini M, De Ponti R, Salerno-Uriarte J A

机构信息

Cardiology Department Mater Domini, Castellanza, Varese, Italy.

出版信息

J Cardiovasc Electrophysiol. 2001 Oct;12(10):1187-9. doi: 10.1046/j.1540-8167.2001.01187.x.

Abstract

A case of iterative atrial tachycardia leading to dilated cardiomyopathy is reported. During electrophysiologic study, the tachycardia showed a markedly irregular cycle length associated with changes in atrial activation breakthrough as demonstrated by coronary sinus (CS) recordings and frequently degenerated into self-terminating atrial fibrillation. Left atrial transseptal mapping demonstrated the earliest endocardial atrial activation close to the posterolateral mitral annulus, but this was invariably later than that recorded within the CS, where low-energy radiofrequency applications eliminated the tachycardia. No acute vessel damage was observed at postablation CS angiography. In accordance with previously published experimental data, we hypothesized that the muscular sleeves surrounding the CS might be involved in the genesis of this tachycardia. During 6-month follow-up, the patient remained asymptomatic without tachycardia recurrences and with complete recovery of left ventricular function, confirming the reversible nature of the tachycardia-induced cardiomyopathy.

摘要

报告了一例反复性房性心动过速导致扩张型心肌病的病例。在电生理研究期间,心动过速显示出明显不规则的周期长度,与冠状窦(CS)记录所显示的心房激动突破变化相关,并且频繁退化为自行终止的心房颤动。左心房经房间隔标测显示最早的心内膜心房激动靠近二尖瓣后外侧瓣环,但这总是晚于CS内记录到的激动,在CS内进行低能量射频消融消除了心动过速。消融后CS血管造影未观察到急性血管损伤。根据先前发表的实验数据,我们推测围绕CS的肌袖可能参与了这种心动过速的发生。在6个月的随访期间,患者无症状,无心动过速复发,左心室功能完全恢复,证实了心动过速性心肌病的可逆性。

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