Krespi Y, Aykutlu E, Coban O, Tunçay R, Bahar S
Department of Neurology, Edip Aktin Stroke Unit, Istanbul School of Medicine, University of Istanbul, Capa, Istanbul, Turkey.
Cerebrovasc Dis. 2001;12(4):346-8. doi: 10.1159/000047733.
Deep hemispheric or brainstem small infarcts can lead to atypical lacunar syndromes. Unilateral internuclear ophthalmoplegia (INO) and cerebellar ataxia has not been reported previously. A 57-year-old hypertensive female presented with bilateral appendicular and left truncal cerebellar ataxia and right INO. Cranial MRI showed a right paramedian infarct of lacunar size located in the tegmentum of caudal mesencephalon. At this level the involvement of medial longitudinal fascicle (MLF) led to right INO and the lesion of brachium conjunctivum caused the bilateral cerebellar ataxia. Ipsilateral involvement of both cerebellofugal fibers, before and after decussation, was responsible for bilateral cerebellar ataxia.
大脑半球深部或脑干小梗死可导致非典型腔隙综合征。单侧核间性眼肌麻痹(INO)和小脑共济失调此前尚未见报道。一名57岁的高血压女性患者出现双侧肢体及左侧躯干性小脑共济失调和右侧INO。头颅MRI显示在尾侧中脑被盖部有一个腔隙大小的右侧旁正中梗死灶。在此层面,内侧纵束(MLF)受累导致右侧INO,结合臂病变引起双侧小脑共济失调。双侧小脑传出纤维在交叉前后的同侧受累是双侧小脑共济失调的原因。