Parr D R, Wimhurst J M, Harris E J
Cardiovasc Res. 1975 May;9(3):366-72. doi: 10.1093/cvr/9.3.366.
The damage which may be caused to heart mitochondria by the rapid uptake of calcium when oxygen is restored after a period of anoxia is monitored by the deterioration in phosphorylation performance. Methods of protecting mitochondria from this damage by preventing calcium uptake and by chelating cytoplasmic calcium are considered.
当缺氧一段时间后恢复氧气供应时，快速摄取钙可能对心脏线粒体造成的损伤，通过磷酸化性能的恶化来监测。人们考虑了通过阻止钙摄取和螯合细胞质钙来保护线粒体免受这种损伤的方法。
