Della Sala S, Francescani A, Spinnler H
Neuropsychology Research Group, Department of Psychology, University of Aberdeen, UK.
J Neurol Neurosurg Psychiatry. 2002 Jan;72(1):77-85. doi: 10.1136/jnnp.72.1.77.
The study aimed at addressing the issue of the precise nature of gait apraxia and the cerebral dysfunction responsible for it.
The case of a patient, affected by a bilateral infarction limited to a portion of the anterior cerebral artery territory is reported. The patient's ability to walk was formally assessed by means of a new standardised test.
Due to an anomaly within the anterior cerebral artery system, the patient's lesion was centred on the supplementary motor regions of both hemispheres. He presented with clear signs of gait apraxia that could not be accounted for by paresis or other neurological deficits. No signs of any other form of apraxia were detected.
The clinical profile of the patient and the analysis of 49 cases from previous literature suggest that gait apraxia should be considered a clinical entity in its own right and lesions to the supplementary motor areas are responsible for it.
本研究旨在探讨步态失用症的确切性质以及导致其发生的脑功能障碍问题。
报告了一例患者,其双侧梗死局限于大脑前动脉区域的一部分。通过一种新的标准化测试对患者的行走能力进行了正式评估。
由于大脑前动脉系统内的异常,患者的病变集中在双侧半球的辅助运动区。他表现出明显的步态失用症迹象,这不能用轻瘫或其他神经功能缺损来解释。未检测到任何其他形式失用症的迹象。
该患者的临床特征以及对先前文献中49例病例的分析表明,步态失用症本身应被视为一种临床实体,辅助运动区的病变是导致其发生的原因。