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育肥牛瘤胃酸中毒的病理形态学变化

[Pathomorphologic changes in calves with ruminal acidosis for fattening].

作者信息

Dzhurov A

出版信息

Vet Med Nauki. 1975;12(7):61-8.

PMID:1179638
Abstract

Studied were the morphologic changes in the organs of 17 calves for fattening with massive acute and subacute acidosis of the rumen. The disease broke out as the result of an abrupt change in the regime of feeding and tending. The gross observations consisted of hemorrhagic and necrotic foci in the rumen, hemorrhages and hyperemia in the omasum and reticulum, and ulcera in the abomasum. Established was also enlargement of the lymph nodes in the forestomach as well as odemas in the liver and kidneys. Histologically, there were necrotic inflammation of the rumen, serous and fibrinous exudate, leukocyte infiltration, hyphae, thrombuses, and arteritis. The omasum and reticulum presented desquamation of the cutaneous mucosal membrane, and the abomasum--necrotic foci, edema, and leukocyte infiltrations. There was also hepatitis, and fatty infiltration of the kidneys and heart and an activation of the interstitium. Degenerative process and hyperemia were observed in the thyroid gland, and fatty infiltration--in the pancreas. The kidneys, the adrenal glands, and the liver had higher alkalinephosphatase activity.

摘要

对17头育肥牛的器官形态变化进行了研究,这些牛患有严重的急性和亚急性瘤胃酸中毒。该病是由于饲养和护理方式突然改变而爆发的。大体观察结果包括瘤胃内的出血和坏死灶、瓣胃和网胃内的出血和充血以及皱胃内的溃疡。还发现前胃淋巴结肿大以及肝脏和肾脏出现水肿。组织学上,瘤胃有坏死性炎症、浆液性和纤维蛋白性渗出物、白细胞浸润、菌丝、血栓和动脉炎。瓣胃和网胃表现为皮肤黏膜脱落,皱胃有坏死灶、水肿和白细胞浸润。还存在肝炎,肾脏、心脏有脂肪浸润以及间质活化。甲状腺出现退行性变和充血,胰腺有脂肪浸润。肾脏、肾上腺和肝脏的碱性磷酸酶活性较高。

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