Castellanos A, Befeler B, Agha A S, Myerburg R J
Arch Inst Cardiol Mex. 1975 Sep-Oct;45(5):655-67.
The analysis of the common bundle and of the ventrical filtered electrograms has incrased our knowledge of the intraventricular conduction following the simulation of the PVS as well as the different types or reciprocating that can be produced by premature beats. 2. The positive QRS complexes in V1 produced from PVD by St2 can be explained as a greater degree of incomplete recuperation of the right ventricular wall than in the interventricular septum. In contrast with what happened in the basal beats, where the front took longer to reach the TSVD than the VI. 3. The refractarines of the transitional tissues that serve as the phisiological banier was longer than the banal myocardium. The intraventricular propagation, and therefore the morphology and direction of QRS, depended of this one; while the moment of arrival of activation retrogradly to the common bundle was conditioned by the transitional tissues. 4. Episodes of ventricular taquicardies with QRS positive complexes in V1 can be due to a re-entry throught the bundle branches or in (or around) an area that has been recently injured.
对共同束和心室滤波心电图的分析，增加了我们对模拟室性早搏后心室传导以及早搏可能产生的不同类型折返的认识。2. 由St2引起的室性早搏在V1导联产生的正向QRS波群，可解释为右心室壁的不完全恢复程度大于室间隔。与基础搏动不同，基础搏动时激动到达室间隔旁道的时间比到达V1的时间长。3. 作为生理屏障的移行组织的不应期比普通心肌长。心室传导，进而QRS波的形态和方向，取决于此；而激动逆向到达共同束的时间则受移行组织的影响。4. V1导联QRS波群正向的室性心动过速发作，可能是由于通过束支或在（或围绕）最近受损区域的折返所致。
