For further investigations in IgA nephropathy the approach from phenotype to genotype is welcome.
作者信息
Schena F P
出版信息
Clin Exp Immunol. 2002 Mar;127(3):399-401. doi: 10.1046/j.1365-2249.2002.01821.x.
Abstract
摘要
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For further investigations in IgA nephropathy the approach from phenotype to genotype is welcome.对于IgA肾病的进一步研究，从表型到基因型的研究方法是值得欢迎的。
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本文引用的文献
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Expression of T cell receptor variable region families by bone marrow gammadelta T cells in patients with IgA nephropathy.IgA肾病患者骨髓γδT细胞中T细胞受体可变区家族的表达
Clin Exp Immunol. 2002 Mar;127(3):527-32. doi: 10.1111/j.1365-2249.2002.01784.x.
2
Oligoclonally expanding gammadelta T lymphocytes induce IgA switching in IgA nephropathy.寡克隆扩增的γδ T淋巴细胞在IgA肾病中诱导IgA类别转换。
Clin Exp Immunol. 2001 Apr;124(1):110-7. doi: 10.1046/j.1365-2249.2001.01494.x.
3
IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.IgA肾病是肾小球肾炎最常见的病因，与6q22 - 23相关。
Nat Genet. 2000 Nov;26(3):354-7. doi: 10.1038/81677.
4
Evidence for genetic factors in the development and progression of IgA nephropathy.IgA肾病发生发展过程中遗传因素的证据。
Kidney Int. 2000 May;57(5):1818-35. doi: 10.1046/j.1523-1755.2000.00032.x.
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Generation of human gammadelta T-cell repertoires.人类γδ T细胞库的生成。
Crit Rev Immunol. 1999;19(5-6):431-60.
6
Expression of the mucosal gamma delta T cell receptor V region repertoire in patients with IgA nephropathy.IgA肾病患者黏膜γδT细胞受体V区库的表达
Kidney Int. 1997 Oct;52(4):1047-53. doi: 10.1038/ki.1997.427.
7
Studies of the molecular basis of IgA production, subclass regulation and class-switch recombination in IgA nephropathy patients.IgA肾病患者中IgA产生、亚类调节和类别转换重组的分子基础研究。
Clin Exp Immunol. 1996 Dec;106(3):509-17. doi: 10.1046/j.1365-2249.1996.d01-859.x.
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The TDT and other family-based tests for linkage disequilibrium and association.传递不平衡检验（TDT）及其他基于家系的连锁不平衡和关联检验。
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Clin Exp Immunol. 1994 Apr;96(1):116-21. doi: 10.1111/j.1365-2249.1994.tb06240.x.
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