Gallenkamp U
Arch Psychiatr Nervenkr (1970). 1979;227(2):135-42.
The occurrence of cerebral seizures in alcoholics was investigated in case histories of 84 delirious and nondelirious male patients. Eighteen patients had seizures before they became alcoholics; the frequency of the seizures increased during abuse. Twelve had no deliria at all up to the moment of this investigation; in 66 of the patients the occurrence of seizures was assumed to be caused by alcohol abuse alone. Seventy-one patients had seizures irrespective of deliria and nearly 40% of them had no deliria at all. In 21% we observed only deliria with seizures; in 16% only deliria without seizures; and 24% had deliria both with and without seizures. The remaining 13 patients of 84 had only deliria complicated by cerebral seizures; only 3 had deliria without seizures. The seizures occurred as grand mal in 94% of the alcoholics, in all patients with genuine epilepsy, and in 60% of the patients with post-traumatic epilepsy.
在84名谵妄和非谵妄男性患者的病历中,对酗酒者癫痫发作的情况进行了调查。18名患者在成为酗酒者之前就有癫痫发作;在酗酒期间癫痫发作的频率增加。12名患者在本次调查时根本没有谵妄;在66名患者中,癫痫发作被认为完全是由酗酒引起的。71名患者无论是否有谵妄都会发作癫痫,其中近40%的患者根本没有谵妄。在21%的患者中,我们观察到仅有伴有癫痫发作的谵妄;在16%的患者中,仅有不伴有癫痫发作的谵妄;24%的患者既有伴有癫痫发作的谵妄,也有不伴有癫痫发作的谵妄。84名患者中其余13名仅有伴有癫痫发作的谵妄;只有3名患者有不伴有癫痫发作的谵妄。94%的酗酒者、所有真正患有癫痫的患者以及60%的创伤后癫痫患者的癫痫发作表现为大发作。