Bader G, Rohde E, Unger M, Koepcke E
Zentralbl Gynakol. 1975;97(21):1317-22.
121 out of 390 placentas of mostly pathological deliveries and preganancies were cases of chorioamnionitis. Histological studies have been performed under topographical respects. Several localisations (dynamic phases) of ascending infection of the secundinae are being described and their clinical relevance is being assessed. 1)"Nomal secundinae" or "physiological leucocytosis at ruptured chorionic membranes": there are but a few cases (3 to 5%) of amniotic infection syndroms or morphological signs of an aspiration of infected amniotic fluid and fetal sepsis. 2) "Isolated leucocytosis of the vessels of the umbilical cord and the chorionic plate": it is mostly caused by a fetal hypoxia; relatively seldom it is the result of an infection (about 10%). 3) "Partial phlegmon of the secundinae" (phlegmon of the chorionic membrane with spreading to the periphery of the chorionic plate): about 30% amniotic infection syndrom or infected amniotic fluid (and fetal sepsis respectively. 4) "Subtotal phlegmon of the secundinae" (phlegmon of the chorionic membrane and the chorionic plate in part, spreading to the umbilical cord): about 50% amniotic infection syndrom or infected amniotic fluid (and fetal sepsis) respectively. 5) "Total phlegmon of the secundinae" : in the majority of cases (about 65%) signs of infection damage on mother and/or fetus are visible.
在390例大多为病理分娩和妊娠的胎盘病例中，有121例发生绒毛膜羊膜炎。已从局部解剖学角度进行了组织学研究。描述了胎膜上行感染的几个部位（动态阶段），并评估了它们的临床相关性。1）“正常胎膜”或“绒毛膜破裂时的生理性白细胞增多”：羊水感染综合征或吸入感染羊水及胎儿败血症的形态学迹象的病例很少（3%至5%）。2）“脐带和绒毛膜板血管孤立性白细胞增多”：主要由胎儿缺氧引起；相对较少是感染的结果（约10%）。3）“胎膜局部蜂窝织炎”（绒毛膜蜂窝织炎并蔓延至绒毛膜板周边）：约30%有羊水感染综合征或感染羊水（及分别有胎儿败血症）。4）“胎膜大部蜂窝织炎”（部分绒毛膜和绒毛膜板蜂窝织炎，蔓延至脐带）：约50%有羊水感染综合征或感染羊水（及胎儿败血症）。5）“胎膜全蜂窝织炎”：在大多数病例中（约65%）可见对母亲和/或胎儿的感染损害迹象。
