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局灶性结节性增生导致肝静脉阻塞。

Focal nodular hyperplasia inducing hepatic vein obstruction.

作者信息

Rangheard Anne-Sophie, Vilgrain Valérie, Audet Pascale, O'Toole Dermot, Vullierme Marie-Pierre, Valla Dominique, Belghiti Jacques, Menu Yves

机构信息

Department of Radiology, Hospital Beaujon, 100 ave. du Général Leclerc, 92110 Clichy, France.

出版信息

AJR Am J Roentgenol. 2002 Sep;179(3):759-62. doi: 10.2214/ajr.179.3.1790759.

DOI:10.2214/ajr.179.3.1790759
PMID:12185058
Abstract

OBJECTIVE

The records of 10 patients with focal nodular hyperplasia inducing intrahepatic vein obstruction were reviewed. The purpose of this study was to describe and emphasize the imaging features of these findings.

CONCLUSION

Focal nodular hyperplasia may be responsible for hepatic vein obstruction with hepatic vein collaterals. The relatively large size and central location of the lesions seem to play important roles in the obstruction of the hepatic veins.

摘要

目的

回顾10例导致肝内静脉阻塞的局灶性结节性增生患者的病历。本研究的目的是描述并强调这些发现的影像学特征。

结论

局灶性结节性增生可能导致肝静脉阻塞并伴有肝静脉侧支循环形成。病变相对较大的尺寸及位于中央的位置似乎在肝静脉阻塞中起重要作用。

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引用本文的文献

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Focal Nodular Hyperplasia: A Comprehensive Review with a Particular Focus on Pathogenesis and Complications.局灶性结节性增生:一项特别关注发病机制和并发症的全面综述
J Clin Transl Hepatol. 2024 Feb 28;12(2):182-190. doi: 10.14218/JCTH.2023.00265. Epub 2024 Jan 2.
2
Giant focal nodular hyperplasia determining Budd-Chiari syndrome: an operative challenge requiring 210 min of liver ischemia.巨块型局灶性结节性增生导致布加综合征:手术面临挑战,需要肝脏缺血 210 分钟。
Updates Surg. 2011 Dec;63(4):307-11. doi: 10.1007/s13304-011-0105-4. Epub 2011 Sep 16.
3
Vascular disorders of the liver.
肝脏血管疾病
Hepatology. 2009 May;49(5):1729-64. doi: 10.1002/hep.22772.
4
Focal nodular hyperplasia: what are the indications for resection?局灶性结节性增生:哪些情况下需要进行切除?
HPB (Oxford). 2005;7(4):298-302. doi: 10.1080/13651820500273624.
5
Angiopoietin-1 causes reversible degradation of the portal microcirculation in mice: implications for treatment of liver disease.血管生成素-1导致小鼠门静脉微循环的可逆性退化:对肝病治疗的启示。
Am J Pathol. 2004 Sep;165(3):889-99. doi: 10.1016/S0002-9440(10)63351-2.