al-Lami F, Farman N
Anat Rec. 1975 Jan;181(1):113-129. doi: 10.1002/ar.1091810109.
The effect of 24 and 48 hours' cold stress on the hamsters' adrenomedullary follicles and on the medullary ATPase activity was studied by light and electron microscopy. Only norepinephrine cells were depleted after this stress, and exocytosis seemed to be the mechanism involved in the release of catecholamine. Follicles containing these cells expanded and their lumina became narrow. A few other cellular and follicular changes also occurred and are described. ATPase activity was apparent in control organs along the endothelial linings, in neural elements and macrophages, and in approximately 40% of the linings of follicular lumina. Cold stress did not alter this pattern. These results have been compared with previous findings and the possible functions of the follicular lumina are discussed. It is concluded that they are unlikely sites for catecholamine storage or release.
通过光学显微镜和电子显微镜研究了24小时和48小时冷应激对仓鼠肾上腺髓质滤泡以及髓质ATP酶活性的影响。应激后仅去甲肾上腺素细胞减少，胞吐作用似乎是儿茶酚胺释放的机制。含有这些细胞的滤泡扩张，其管腔变窄。还发生了一些其他细胞和滤泡变化并进行了描述。在对照器官中，ATP酶活性在内皮衬里、神经成分和巨噬细胞中明显，并且在约40%的滤泡管腔衬里中也有活性。冷应激并未改变这种模式。将这些结果与先前的发现进行了比较，并讨论了滤泡管腔的可能功能。得出的结论是，它们不太可能是儿茶酚胺储存或释放的部位。
