α-突触核蛋白与突触前功能：对帕金森病的影响
Alpha-synuclein and presynaptic function: implications for Parkinson's disease.
作者信息
Lykkebo Simon, Jensen Poul Henning
机构信息
Department of Medical Biochemistry, Aarhus University, Denmark.
出版信息
Neuromolecular Med. 2002;2(2):115-29. doi: 10.1385/NMM:2:2:115.
PMID:12428807
Abstract
This article focuses on alpha-synuclein's role in normal and pathological axonal and presynaptic functions and its relationship to Parkinson's disease. It is not possible to mention all the contributions to aspects of this area. Readers interested in alpha-synuclein's relation to aggregation, Lewy lesions, and pathological modifications are referred to the many reviews (see Goldberg and Lansbury 2000; Galvin 2001a; Goedert 2001).
摘要
本文重点关注α-突触核蛋白在正常和病理性轴突及突触前功能中的作用及其与帕金森病的关系。不可能提及该领域所有方面的贡献。对α-突触核蛋白与聚集、路易小体及病理改变关系感兴趣的读者可参考众多综述（见Goldberg和Lansbury，2000年；Galvin，2001年a；Goedert，2001年）。
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