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The mechanism of formation of pulmonary arteriovenous malformations associated with the classic Glenn shunt (superior cavopulmonary anastomosis).

作者信息

Ashrafian H, Swan L

出版信息

Heart. 2002 Dec;88(6):639. doi: 10.1136/heart.88.6.639.

DOI:10.1136/heart.88.6.639
PMID:12433901
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1767473/
Abstract
摘要

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本文引用的文献

1
Reduced hypoxic pulmonary vascular remodeling by nitric oxide from the endothelium.内皮细胞产生的一氧化氮减少低氧性肺血管重塑。
Hypertension. 2001 Feb;37(2):322-7. doi: 10.1161/01.hyp.37.2.322.
2
Diagnostic criteria for hereditary hemorrhagic telangiectasia (Rendu-Osler-Weber syndrome).遗传性出血性毛细血管扩张症（伦杜-奥斯勒-韦伯综合征）的诊断标准。
Am J Med Genet. 2000 Mar 6;91(1):66-7. doi: 10.1002/(sici)1096-8628(20000306)91:1<66::aid-ajmg12>3.0.co;2-p.
3
Reversal of pulmonary arteriovenous malformation after diversion of anomalous hepatic drainage.异常肝引流改道后肺动静脉畸形的逆转
Ann Thorac Surg. 1998 Mar;65(3):848-9. doi: 10.1016/s0003-4975(98)00011-3.
4
Endothelial cells inhibit NO generation by vascular smooth muscle cells. Role of transforming growth factor-beta.
Arterioscler Thromb Vasc Biol. 1996 Oct;16(10):1263-8. doi: 10.1161/01.atv.16.10.1263.
5
Hepatic venous blood and the development of pulmonary arteriovenous malformations in congenital heart disease.先天性心脏病中肝静脉血与肺动静脉畸形的发展
Circulation. 1995 Sep 1;92(5):1217-22. doi: 10.1161/01.cir.92.5.1217.