Baier V, Leder U, Voss A
Fachbereich Medizintechnik, Fachhochschule Jena, Deutschland.
Biomed Tech (Berl). 2002;47 Suppl 1 Pt 2:871-4. doi: 10.1515/bmte.2002.47.s1b.871.
Noninvasive finger arterial blood pressure and ECG were recorded for 30 min in patients with idiopathic dilated cardiomyopathy (IDC) and healthy control subjects (CON) for the investigation of blood pressure and heart rate regulation following a premature ventricular complex (PVC). The potentiation of blood pressure amplitude of the postextrasystolic beat was fivefold increased in IDC compared with CON (48.7 +/- 32.6% vs. 9.8 +/- 5.4%, p < 0.01). PVC starts a baroreflex response, which last about 10 s in physiological regulation. In contrast, the baroreflex response in IDC is immediately suppressed by the augmented potentiation of the blood pressure amplitude from the first postextrasystolic beat. Thus, the regulation after PVC is determined by the PVC itself and postextrasystic potentiation.
对特发性扩张型心肌病(IDC)患者和健康对照者(CON)进行30分钟的无创手指动脉血压和心电图记录,以研究室性早搏(PVC)后血压和心率的调节情况。与CON相比,IDC患者早搏后搏动的血压振幅增强增加了五倍(48.7±32.6%对9.8±5.4%,p<0.01)。PVC引发压力反射反应,在生理调节中持续约10秒。相比之下,IDC患者的压力反射反应在早搏后第一个搏动的血压振幅增强时立即受到抑制。因此,PVC后的调节由PVC本身和早搏后增强作用决定。