Swarup Vijendra, Morton Joseph B, Arruda Mauricio, Wilber David J
Loyola University Medical Center, Maywood, Illinois 60153, USA.
J Cardiovasc Electrophysiol. 2002 Nov;13(11):1164-8. doi: 10.1046/j.1540-8167.2002.01164.x.
Characterization of the substrate and mechanism of epicardial ventricular tachycardia (VT) associated with idiopathic nonischemic dilated cardiomyopathy is limited. We report a case of successful mapping and ablation of an epicardial VT by a percutaneous transthoracic approach in a patient with idiopathic dilated cardiomyopathy, frequent VT, and previously unsuccessful endocardial ablation. Evidence of myocardial scar was limited to the epicardium. Electroanatomic and entrainment mapping defined a figure-of-eight macroreentrant circuit within the epicardial scar. VT terminated at the onset of low-power radiofrequency application to the central isthmus of the circuit. VT was no longer induced and did not recur during long-term follow-up.
与特发性非缺血性扩张型心肌病相关的心外膜室性心动过速(VT)的底物和机制的特征尚不明确。我们报告了1例特发性扩张型心肌病、频发室性心动过速且既往心内膜消融失败的患者,经皮经胸途径成功标测并消融心外膜室性心动过速的病例。心肌瘢痕的证据仅限于心外膜。电解剖和拖带标测确定了心外膜瘢痕内的8字形大折返环。在对环路中央峡部施加低功率射频时,室性心动过速终止。室性心动过速不再被诱发,且在长期随访中未复发。